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Acquired hyperkalaemia leading to periodic paralysis: an emergency department perspective.
- Source :
-
BMJ case reports [BMJ Case Rep] 2024 May 09; Vol. 17 (5). Date of Electronic Publication: 2024 May 09. - Publication Year :
- 2024
-
Abstract
- Hyperkalaemia is one of the common electrolyte imbalances dealt with in the emergency department and is caused by extracellular accumulation of potassium ions above normal limits usually greater than 5.0-5.5 mmol/L. It is found in a total of 1-10% of hospitalised patients usually associated with chronic kidney disease and heart failure. The presentation can range from being asymptomatic to deadly arrhythmias. The appearance of symptoms depends on the rate of change rather than just the numerical values. The rare presentation includes periodic paralysis characterised by the sudden onset of short-term muscle weakness, stiffness or paralysis. Management goals are directed towards reducing potassium levels in emergency settings and later on avoiding the triggers for future attacks. In this case, we present a man in his 50s with the generalised weakness later on diagnosed as hyperkalaemic periodic paralysis secondary to tumour lysis syndrome. Emergency physicians dealing with common electrolyte imbalances should keep a sharp eye on their rare presentation and their precipitating factors and should act accordingly.<br />Competing Interests: Competing interests: None declared.<br /> (© BMJ Publishing Group Limited 2024. No commercial re-use. See rights and permissions. Published by BMJ.)
- Subjects :
- Humans
Male
Middle Aged
Paralysis, Hyperkalemic Periodic diagnosis
Paralysis, Hyperkalemic Periodic complications
Potassium blood
Potassium therapeutic use
Diagnosis, Differential
Muscle Weakness etiology
Emergency Service, Hospital
Hyperkalemia etiology
Hyperkalemia diagnosis
Hyperkalemia therapy
Subjects
Details
- Language :
- English
- ISSN :
- 1757-790X
- Volume :
- 17
- Issue :
- 5
- Database :
- MEDLINE
- Journal :
- BMJ case reports
- Publication Type :
- Academic Journal
- Accession number :
- 38724210
- Full Text :
- https://doi.org/10.1136/bcr-2024-260151