The Compromised Fanconi Anemia Pathway in Prelamin A-Expressing Cells Contributes to Replication Stress-Induced Genomic Instability.

MLA

Nie, Pengqing, et al. “The Compromised Fanconi Anemia Pathway in Prelamin A-Expressing Cells Contributes to Replication Stress-Induced Genomic Instability.” Advanced Science (Weinheim, Baden-Wurttemberg, Germany), vol. 11, no. 30, Aug. 2024, p. e2307751. EBSCOhost, https://doi.org/10.1002/advs.202307751.

APA

Nie, P., Zhang, C., Wu, F., Chen, S., & Wang, L. (2024). The Compromised Fanconi Anemia Pathway in Prelamin A-Expressing Cells Contributes to Replication Stress-Induced Genomic Instability. Advanced Science (Weinheim, Baden-Wurttemberg, Germany), 11(30), e2307751. https://doi.org/10.1002/advs.202307751

Chicago

Nie, Pengqing, Cheng Zhang, Fengyi Wu, Shi Chen, and Lianrong Wang. 2024. “The Compromised Fanconi Anemia Pathway in Prelamin A-Expressing Cells Contributes to Replication Stress-Induced Genomic Instability.” Advanced Science (Weinheim, Baden-Wurttemberg, Germany) 11 (30): e2307751. doi:10.1002/advs.202307751.