Nesfatin-1 regulates the phenotype transition of cavernous smooth muscle cells by activating PI3K/AKT/mTOR signaling pathway to improve diabetic erectile dysfunction.

MLA

Chen, Keming, et al. “Nesfatin-1 Regulates the Phenotype Transition of Cavernous Smooth Muscle Cells by Activating PI3K/AKT/MTOR Signaling Pathway to Improve Diabetic Erectile Dysfunction.” PloS One, vol. 19, no. 9, Sept. 2024, p. e0304485. EBSCOhost, https://doi.org/10.1371/journal.pone.0304485.

APA

Chen, K., Huang, B., Feng, J., Hu, Z., Fan, S., Ren, S., Tian, H., Abdulkarem M M, A.-Q. M., Wang, X., Tuo, Y., Liang, X., Xie, H., He, R., & Li, G. (2024). Nesfatin-1 regulates the phenotype transition of cavernous smooth muscle cells by activating PI3K/AKT/mTOR signaling pathway to improve diabetic erectile dysfunction. PloS One, 19(9), e0304485. https://doi.org/10.1371/journal.pone.0304485

Chicago

Chen, Keming, Bincheng Huang, Jiajing Feng, Zhengxing Hu, Shuzhe Fan, Shuai Ren, Haifu Tian, et al. 2024. “Nesfatin-1 Regulates the Phenotype Transition of Cavernous Smooth Muscle Cells by Activating PI3K/AKT/MTOR Signaling Pathway to Improve Diabetic Erectile Dysfunction.” PloS One 19 (9): e0304485. doi:10.1371/journal.pone.0304485.