Cite
USP20 deletion promotes eccentric cardiac remodeling in response to pressure overload and increases mortality.
MLA
Jean-Charles, Pierre-Yves, et al. “USP20 Deletion Promotes Eccentric Cardiac Remodeling in Response to Pressure Overload and Increases Mortality.” American Journal of Physiology. Heart and Circulatory Physiology, vol. 327, no. 5, Nov. 2024, pp. H1257–71. EBSCOhost, https://doi.org/10.1152/ajpheart.00329.2024.
APA
Jean-Charles, P.-Y., Roy, B., Yu, S. M.-W., Pironti, G., Nagi, K., Mao, L., Kaur, S., Abraham, D. M., Maudsley, S., Rockman, H. A., & Shenoy, S. K. (2024). USP20 deletion promotes eccentric cardiac remodeling in response to pressure overload and increases mortality. American Journal of Physiology. Heart and Circulatory Physiology, 327(5), H1257–H1271. https://doi.org/10.1152/ajpheart.00329.2024
Chicago
Jean-Charles, Pierre-Yves, Bipradas Roy, Samuel Mon-Wei Yu, Gianluigi Pironti, Karim Nagi, Lan Mao, Suneet Kaur, et al. 2024. “USP20 Deletion Promotes Eccentric Cardiac Remodeling in Response to Pressure Overload and Increases Mortality.” American Journal of Physiology. Heart and Circulatory Physiology 327 (5): H1257–71. doi:10.1152/ajpheart.00329.2024.