tp53 R217H and R242H mutant zebrafish exhibit dysfunctional p53 hallmarks and recapitulate Li-Fraumeni syndrome phenotypes.

MLA

Kobar, Kim, et al. “Tp53 R217H and R242H Mutant Zebrafish Exhibit Dysfunctional P53 Hallmarks and Recapitulate Li-Fraumeni Syndrome Phenotypes.” Biochimica et Biophysica Acta. Molecular Basis of Disease, vol. 1871, no. 3, Dec. 2024, p. 167612. EBSCOhost, https://doi.org/10.1016/j.bbadis.2024.167612.

APA

Kobar, K., Tuzi, L., Fiene, J. A., Burnley, E., Galpin, K. J. C., Midgen, C., Laverty, B., Subasri, V., Wen, T. T., Hirst, M., Moksa, M., Carles, A., Cao, Q., Shlien, A., Malkin, D., Prykhozhij, S. V., & Berman, J. N. (2024). tp53 R217H and R242H mutant zebrafish exhibit dysfunctional p53 hallmarks and recapitulate Li-Fraumeni syndrome phenotypes. Biochimica et Biophysica Acta. Molecular Basis of Disease, 1871(3), 167612. https://doi.org/10.1016/j.bbadis.2024.167612

Chicago

Kobar, Kim, Lissandra Tuzi, Jennifer A Fiene, Erin Burnley, Kristianne J C Galpin, Craig Midgen, Brianne Laverty, et al. 2024. “Tp53 R217H and R242H Mutant Zebrafish Exhibit Dysfunctional P53 Hallmarks and Recapitulate Li-Fraumeni Syndrome Phenotypes.” Biochimica et Biophysica Acta. Molecular Basis of Disease 1871 (3): 167612. doi:10.1016/j.bbadis.2024.167612.