Propofol attenuates HO-induced oxidative stress and apoptosis via the mitochondria- and ER-medicated pathways in neonatal rat cardiomyocytes.

MLA

Liu, Xue-Ru, et al. “Propofol Attenuates HO-Induced Oxidative Stress and Apoptosis via the Mitochondria- and ER-Medicated Pathways in Neonatal Rat Cardiomyocytes.” Apoptosis, vol. 22, no. 5, May 2017, pp. 639–46. EBSCOhost, https://doi.org/10.1007/s10495-017-1349-3.

APA

Liu, X.-R., Cao, L., Li, T., Chen, L.-L., Yu, Y.-Y., Huang, W.-J., Liu, L., & Tan, X.-Q. (2017). Propofol attenuates HO-induced oxidative stress and apoptosis via the mitochondria- and ER-medicated pathways in neonatal rat cardiomyocytes. Apoptosis, 22(5), 639–646. https://doi.org/10.1007/s10495-017-1349-3

Chicago

Liu, Xue-Ru, Lu Cao, Tao Li, Lin-Lin Chen, Yi-Yan Yu, Wen-Jun Huang, Li Liu, and Xiao-Qiu Tan. 2017. “Propofol Attenuates HO-Induced Oxidative Stress and Apoptosis via the Mitochondria- and ER-Medicated Pathways in Neonatal Rat Cardiomyocytes.” Apoptosis 22 (5): 639–46. doi:10.1007/s10495-017-1349-3.