Neuronal susceptibility to beta‐amyloid toxicity and ischemic injury involves histone deacetylase‐2 regulation of endophilin‐B1.

MLA

Wang, David B., et al. “Neuronal Susceptibility to Beta‐amyloid Toxicity and Ischemic Injury Involves Histone Deacetylase‐2 Regulation of Endophilin‐B1.” Brain Pathology, vol. 29, no. 2, Mar. 2019, pp. 164–75. EBSCOhost, https://doi.org/10.1111/bpa.12647.

APA

Wang, D. B., Kinoshita, C., Kinoshita, Y., Sopher, B. L., Uo, T., Lee, R. J., Kim, J. K., Murphy, S. P., Dirk Keene, C., Garden, G. A., & Morrison, R. S. (2019). Neuronal susceptibility to beta‐amyloid toxicity and ischemic injury involves histone deacetylase‐2 regulation of endophilin‐B1. Brain Pathology, 29(2), 164–175. https://doi.org/10.1111/bpa.12647

Chicago

Wang, David B., Chizuru Kinoshita, Yoshito Kinoshita, Bryce L. Sopher, Takuma Uo, Rona J. Lee, Joon Kyu Kim, et al. 2019. “Neuronal Susceptibility to Beta‐amyloid Toxicity and Ischemic Injury Involves Histone Deacetylase‐2 Regulation of Endophilin‐B1.” Brain Pathology 29 (2): 164–75. doi:10.1111/bpa.12647.