Autoimmune Cerebellar Ataxia due to Inflammation of the Inferior Olivary Nuclei.

Impaired tilt suppression of post-rotatory nystagmus and cross-coupled head-shaking nystagmus in cerebellar lesions: image mapping study. Lesions involving the ION have mostly been reported as a form of pseudo-hypertrophic olivary degeneration (HOD) that refers to vacuolar degeneration of the enlarged neurons in the ION and mostly develops several months after damage to the dentato-rubro-olivary tract (the Guillain-Mollaret triangle) [[2]]. The inferior olivary nucleus (ION) receives signals from the spinal cord and cerebellum, and regulates coordination and motor learning [[1]]. [Extracted from the article]