Inhibition of the H3K27 demethylase UTX enhances the epigenetic silencing of HIV proviruses and induces HIV-1 DNA hypermethylation but fails to permanently block HIV reactivation.

MLA

Nguyen, Kien, et al. “Inhibition of the H3K27 Demethylase UTX Enhances the Epigenetic Silencing of HIV Proviruses and Induces HIV-1 DNA Hypermethylation but Fails to Permanently Block HIV Reactivation.” PLoS Pathogens, vol. 17, no. 10, Oct. 2021, pp. 1–30. EBSCOhost, https://doi.org/10.1371/journal.ppat.1010014.

APA

Nguyen, K., Dobrowolski, C., Shukla, M., Cho, W.-K., Luttge, B., & Karn, J. (2021). Inhibition of the H3K27 demethylase UTX enhances the epigenetic silencing of HIV proviruses and induces HIV-1 DNA hypermethylation but fails to permanently block HIV reactivation. PLoS Pathogens, 17(10), 1–30. https://doi.org/10.1371/journal.ppat.1010014

Chicago

Nguyen, Kien, Curtis Dobrowolski, Meenakshi Shukla, Won-Kyung Cho, Benjamin Luttge, and Jonathan Karn. 2021. “Inhibition of the H3K27 Demethylase UTX Enhances the Epigenetic Silencing of HIV Proviruses and Induces HIV-1 DNA Hypermethylation but Fails to Permanently Block HIV Reactivation.” PLoS Pathogens 17 (10): 1–30. doi:10.1371/journal.ppat.1010014.