Research on Reperfusion Injury Discussed by Researchers at University of Chile (Cardioprotective Mechanisms against Reperfusion Injury in Acute Myocardial Infarction: Targeting Angiotensin II Receptors).

Cardiovascular Diseases and Conditions - Reperfusion Injury Investigators discuss new findings in reperfusion injury. A major cardiovascular ROS source in this setting is theactivation of multiple NADPH oxidases, which could result via theoccupancy of type 1 angiotensin II receptors (AT1R); hence, the reninangiotensin system (RAS) is associated with the generation of ROS duringreperfusion. [Extracted from the article]