Mitochondrial complex I inhibition triggers NAD+-independent glucose oxidation via successive NADPH formation, "futile" fatty acid cycling, and FADH2 oxidation.

MLA

Abrosimov, Roman, et al. “Mitochondrial Complex I Inhibition Triggers NAD+-Independent Glucose Oxidation via Successive NADPH Formation, ‘Futile’ Fatty Acid Cycling, and FADH2 Oxidation.” GeroScience, vol. 46, no. 4, Aug. 2024, pp. 3635–58. EBSCOhost, https://doi.org/10.1007/s11357-023-01059-y.

APA

Abrosimov, R., Baeken, M. W., Hauf, S., Wittig, I., Hajieva, P., Perrone, C. E., & Moosmann, B. (2024). Mitochondrial complex I inhibition triggers NAD+-independent glucose oxidation via successive NADPH formation, “futile” fatty acid cycling, and FADH2 oxidation. GeroScience, 46(4), 3635–3658. https://doi.org/10.1007/s11357-023-01059-y

Chicago

Abrosimov, Roman, Marius W. Baeken, Samuel Hauf, Ilka Wittig, Parvana Hajieva, Carmen E. Perrone, and Bernd Moosmann. 2024. “Mitochondrial Complex I Inhibition Triggers NAD+-Independent Glucose Oxidation via Successive NADPH Formation, ‘Futile’ Fatty Acid Cycling, and FADH2 Oxidation.” GeroScience 46 (4): 3635–58. doi:10.1007/s11357-023-01059-y.