SP1 promotes high glucose-induced lens epithelial cell viability, migration and epithelial-mesenchymal transition via regulating FGF7 and PI3K/AKT pathway.

MLA

Wang, Ledan, et al. “SP1 Promotes High Glucose-Induced Lens Epithelial Cell Viability, Migration and Epithelial-Mesenchymal Transition via Regulating FGF7 and PI3K/AKT Pathway.” International Ophthalmology, July 2024, pp. 1–11. EBSCOhost, https://doi.org/10.1007/s10792-024-03230-6.

APA

Wang, L., Zhang, X., Li, H., Mou, Y., & Cui, G. (2024). SP1 promotes high glucose-induced lens epithelial cell viability, migration and epithelial-mesenchymal transition via regulating FGF7 and PI3K/AKT pathway. International Ophthalmology, 1–11. https://doi.org/10.1007/s10792-024-03230-6

Chicago

Wang, Ledan, Xin Zhang, Huijun Li, Yuehong Mou, and Gangfeng Cui. 2024. “SP1 Promotes High Glucose-Induced Lens Epithelial Cell Viability, Migration and Epithelial-Mesenchymal Transition via Regulating FGF7 and PI3K/AKT Pathway.” International Ophthalmology, July, 1–11. doi:10.1007/s10792-024-03230-6.