B-PO02-032 SUCCINATE ACCUMULATION IN ATRIAL CARDIOMYOCYTES INCREASES MITOCHONDRIAL REVERSE ELECTRON FLUX, OXIDATIVE STRESS, AND MIGHT PARTICIPATE TO AF STABILIZATION IN THE SHEEP.

MLA

Caluori, Guido, et al. “B-Po02-032 Succinate Accumulation in Atrial Cardiomyocytes Increases Mitochondrial Reverse Electron Flux, Oxidative Stress, and Might Participate to Af Stabilization in the Sheep.” Heart Rhythm, vol. 18, no. 8, Aug. 2021, pp. S107–08. EBSCOhost, https://doi.org/10.1016/j.hrthm.2021.06.288.

APA

Caluori, G., Vaillant, F., Abell, E., Ichou, F., Loyer, V., Häberlin, A., Ploux, S., Mahamat, H.-A., Dubois, R., Guillot, B., Diolez, P., Meillet, V., Hatem, S., Krisai, P., Kamakura, T., Hocini, M., Bernus, O., Dos Santos, P., Jais, P., & Pasdois, P. (2021). B-Po02-032 Succinate Accumulation in Atrial Cardiomyocytes Increases Mitochondrial Reverse Electron Flux, Oxidative Stress, and Might Participate to Af Stabilization in the Sheep. Heart Rhythm, 18(8), S107–S108. https://doi.org/10.1016/j.hrthm.2021.06.288

Chicago

Caluori, Guido, Fanny Vaillant, Emma Abell, Farid Ichou, Virginie Loyer, Andreas Häberlin, Sylvain Ploux, et al. 2021. “B-Po02-032 Succinate Accumulation in Atrial Cardiomyocytes Increases Mitochondrial Reverse Electron Flux, Oxidative Stress, and Might Participate to Af Stabilization in the Sheep.” Heart Rhythm 18 (8): S107–8. doi:10.1016/j.hrthm.2021.06.288.