Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy with Features of Left Ventricular Noncompaction

MLA

Hastings R, et al. Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy with Features of Left Ventricular Noncompaction. Oct. 2016. EBSCOhost, https://doi.org/10.1161/CIRCGENETICS.116.001431.

APA

Hastings R, Cp, V., Hooper C, Elizabeth Ormondroyd, Pagnamenta A, Lise S, Salatino S, Sj, K., Jc, T., Kl, T., Arnold L, Sd, C., Pv, K., Wilmanns M, Ehler E, Ghisleni A, Gautel M, Blair E, Watkins H, & Gehmlich K. (2016). Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy with Features of Left Ventricular Noncompaction. https://doi.org/10.1161/CIRCGENETICS.116.001431

Chicago

Hastings R, Villiers Cp, Hooper C, Elizabeth Ormondroyd, Pagnamenta A, Lise S, Salatino S, et al. 2016. “Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy with Features of Left Ventricular Noncompaction,” October. doi:10.1161/CIRCGENETICS.116.001431.