Su1651 Activation of Pancreatic Enzyme Plus Bacterial Infection Plays an Important Role in the Pathogenic Mechanism of Clinically Relevant POPF After Pancreaticoduodenectomy

MLA

Kenichiro Uemura, et al. “Su1651 Activation of Pancreatic Enzyme Plus Bacterial Infection Plays an Important Role in the Pathogenic Mechanism of Clinically Relevant POPF After Pancreaticoduodenectomy.” Gastroenterology, vol. 144, May 2013, p. S-1082. EBSCOhost, https://doi.org/10.1016/s0016-5085(13)64034-2.

APA

Kenichiro Uemura, Taijiro Sueda, Takeshi Sudo, Hayato Sasaki, Yoshiaki Murakami, Hiroki Ohge, Naru Kondo, Kenjiro Okada, Yasushi Hashimoto, & Naoya Nakagawa. (2013). Su1651 Activation of Pancreatic Enzyme Plus Bacterial Infection Plays an Important Role in the Pathogenic Mechanism of Clinically Relevant POPF After Pancreaticoduodenectomy. Gastroenterology, 144, S-1082. https://doi.org/10.1016/s0016-5085(13)64034-2

Chicago

Kenichiro Uemura, Taijiro Sueda, Takeshi Sudo, Hayato Sasaki, Yoshiaki Murakami, Hiroki Ohge, Naru Kondo, Kenjiro Okada, Yasushi Hashimoto, and Naoya Nakagawa. 2013. “Su1651 Activation of Pancreatic Enzyme Plus Bacterial Infection Plays an Important Role in the Pathogenic Mechanism of Clinically Relevant POPF After Pancreaticoduodenectomy.” Gastroenterology 144 (May): S-1082. doi:10.1016/s0016-5085(13)64034-2.