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Molecular Mechanism of Agonism and Inverse Agonism in the Melanocortin Receptors
- Source :
- Annals of the New York Academy of Sciences. 994:1-11
- Publication Year :
- 2003
- Publisher :
- Wiley, 2003.
-
Abstract
- Among the rhodopsin-like 7TM receptors, the MC receptors are functionally unique because their high constitutive signaling activity is regulated not only by endogenous peptide agonists-MSH peptides-but also by endogenous inverse agonists, namely, the proteins agouti and AGRP. Moreover, the metal-ion Zn(2+) increases the signaling activity of at least the MC1 and MC4 receptors in three distinct ways: (1). by directly functioning as an agonist; (2). by potentiating the action of the endogenous agonist; and (3). by inhibiting the binding of the endogenous inverse agonist. Structurally the MC receptors are part of a small subset of 7TM receptors in which the main ligand-binding crevice, and especially extracellular loops 2 and 3, appear to be specially designed for easy ligand access and bias towards an active state of the receptor-i.e., constitutive activity. Thus, in the MC receptors extracellular loop 2 is ultrashort because TM-IV basically connects directly into TM-V, whereas extracellular loop 3 appears to be held in a particular, constrained conformation by a putative, internal disulfide bridge. The interaction mode for the small and well-defined zinc-ion between a third, free Cys residue in extracellular loop 3 and conceivably an Asp residue located at the inner face of TM-III gives important information concerning the activation mechanism for the MC receptors.
- Subjects :
- Agonist
Chemistry
medicine.drug_class
General Neuroscience
General Biochemistry, Genetics and Molecular Biology
History and Philosophy of Science
Biochemistry
medicine
Extracellular
Biophysics
Inverse agonist
Melanocortin
Signal transduction
Receptor
Endogenous agonist
G protein-coupled receptor
Subjects
Details
- ISSN :
- 17496632 and 00778923
- Volume :
- 994
- Database :
- OpenAIRE
- Journal :
- Annals of the New York Academy of Sciences
- Accession number :
- edsair.doi...........76518f6427f1ce9f3e1ded06770be9fe
- Full Text :
- https://doi.org/10.1111/j.1749-6632.2003.tb03156.x