Epigenetic silencing of GCH1promotes hepatocellular carcinoma growth by activating superoxide anion-mediated ASK1/p38 signaling via inhibiting tetrahydrobiopterin de novo biosynthesis

MLA

Dong Cai, et al. “Epigenetic Silencing of GCH1promotes Hepatocellular Carcinoma Growth by Activating Superoxide Anion-Mediated ASK1/P38 Signaling via Inhibiting Tetrahydrobiopterin de Novo Biosynthesis.” Free Radical Biology and Medicine, vol. 168, May 2021, pp. 81–94. EBSCOhost, https://doi.org/10.1016/j.freeradbiomed.2021.03.025.

APA

Dong Cai, Lin-Hong Mao, Chan Qiu, Guo-Chao Zhong, Jianping Gong, Zhibo Zhao, Sheng-Wei Li, Yun-Bing Wang, Jie-Jun Hu, Yao Cheng, & Yan Liu. (2021). Epigenetic silencing of GCH1promotes hepatocellular carcinoma growth by activating superoxide anion-mediated ASK1/p38 signaling via inhibiting tetrahydrobiopterin de novo biosynthesis. Free Radical Biology and Medicine, 168, 81–94. https://doi.org/10.1016/j.freeradbiomed.2021.03.025

Chicago

Dong Cai, Lin-Hong Mao, Chan Qiu, Guo-Chao Zhong, Jianping Gong, Zhibo Zhao, Sheng-Wei Li, et al. 2021. “Epigenetic Silencing of GCH1promotes Hepatocellular Carcinoma Growth by Activating Superoxide Anion-Mediated ASK1/P38 Signaling via Inhibiting Tetrahydrobiopterin de Novo Biosynthesis.” Free Radical Biology and Medicine 168 (May): 81–94. doi:10.1016/j.freeradbiomed.2021.03.025.