Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes

MLA

Joshua A. Sommers, et al. “Fanconi Anemia Group J Mutation Abolishes Its DNA Repair Function by Uncoupling DNA Translocation from Helicase Activity or Disruption of Protein-DNA Complexes.” Blood, vol. 116, Nov. 2010, pp. 3780–91. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....1d0eca3a7515c6edc8dbd52b8360192f&authtype=sso&custid=ns315887.

APA

Joshua A. Sommers, Alexander V. Mazin, Yuliang Wu, Avvaru N. Suhasini, Thomas A. Leonard, Kazuo Shin-ya, Robert M. Brosh, Hiroyuki Kitao, & Julianna S. Deakyne. (2010). Fanconi anemia group J mutation abolishes its DNA repair function by uncoupling DNA translocation from helicase activity or disruption of protein-DNA complexes. Blood, 116, 3780–3791.

Chicago

Joshua A. Sommers, Alexander V. Mazin, Yuliang Wu, Avvaru N. Suhasini, Thomas A. Leonard, Kazuo Shin-ya, Robert M. Brosh, Hiroyuki Kitao, and Julianna S. Deakyne. 2010. “Fanconi Anemia Group J Mutation Abolishes Its DNA Repair Function by Uncoupling DNA Translocation from Helicase Activity or Disruption of Protein-DNA Complexes.” Blood 116 (November): 3780–91. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....1d0eca3a7515c6edc8dbd52b8360192f&authtype=sso&custid=ns315887.