Sustained Interleukin-1β Overexpression Exacerbates Tau Pathology Despite Reduced Amyloid Burden in an Alzheimer's Mouse Model

MLA

Simantini Ghosh, et al. “Sustained Interleukin-1β Overexpression Exacerbates Tau Pathology Despite Reduced Amyloid Burden in an Alzheimer’s Mouse Model.” The Journal of Neuroscience, vol. 33, Mar. 2013, pp. 5053–64. EBSCOhost, https://doi.org/10.1523/jneurosci.4361-12.2013.

APA

Simantini Ghosh, Stephanos Kyrkanides, M. Kerry O’Banion, Michael D. Wu, John A. Olschowka, Frank M. LaFerla, & Solomon S. Shaftel. (2013). Sustained Interleukin-1β Overexpression Exacerbates Tau Pathology Despite Reduced Amyloid Burden in an Alzheimer’s Mouse Model. The Journal of Neuroscience, 33, 5053–5064. https://doi.org/10.1523/jneurosci.4361-12.2013

Chicago

Simantini Ghosh, Stephanos Kyrkanides, M. Kerry O’Banion, Michael D. Wu, John A. Olschowka, Frank M. LaFerla, and Solomon S. Shaftel. 2013. “Sustained Interleukin-1β Overexpression Exacerbates Tau Pathology Despite Reduced Amyloid Burden in an Alzheimer’s Mouse Model.” The Journal of Neuroscience 33 (March): 5053–64. doi:10.1523/jneurosci.4361-12.2013.