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Ultrasensitive quantitative measurement of huntingtin phosphorylation at residue S13
- Source :
- Biochemical and Biophysical Research Communications. 521:549-554
- Publication Year :
- 2020
- Publisher :
- Elsevier BV, 2020.
-
Abstract
- Huntington's disease (HD) is a progressive neurodegenerative disorder caused by an expansion of a CAG triplet repeat (encoding for a polyglutamine tract) within the first exon of the huntingtin gene. Expression of the mutant huntingtin (mHTT) protein can result in the production of N-terminal fragments with a robust propensity to form oligomers and aggregates, which may be causally associated with HD pathology. Several lines of evidence indicate that N17 phosphorylation or pseudophosphorylation at any of the residues T3, S13 or S16, alone or in combination, modulates mHTT aggregation, subcellular localization and toxicity. Consequently, increasing N17 phosphorylation has been proposed as a potential therapeutic approach. However, developing genetic/pharmacological tools to quantify these phosphorylation events is necessary in order to subsequently develop tool modulators, which is difficult given the transient and incompletely penetrant nature of such post-translational modifications. Here we describe the first ultrasensitive sandwich immunoassay that quantifies HTT phosphorylated at residue S13 and demonstrate its utility for specific analyte detection in preclinical models of HD. (C) 2019 Published by Elsevier Inc.
- Subjects :
- exon-1
huntington's disease
Huntingtin
Mutant
Biophysics
Biochemistry
ganglioside gm1
hd mutation
Mice
Protein Aggregates
Exon
Huntington's disease
post-translational modifications
medicine
Animals
Humans
immunoassay
Gene Knock-In Techniques
Phosphorylation
Molecular Biology
Cells, Cultured
Neurons
disease
Huntingtin Protein
Chemistry
pathogenesis
Neurodegeneration
neurodegeneration
mutant huntingtin
aggregation
Cell Biology
Polyglutamine tract
medicine.disease
Subcellular localization
Cell biology
nuclear
HEK293 Cells
Huntington Disease
Mutation
posttranslational modifications
protein
Protein Processing, Post-Translational
Subjects
Details
- ISSN :
- 0006291X
- Volume :
- 521
- Database :
- OpenAIRE
- Journal :
- Biochemical and Biophysical Research Communications
- Accession number :
- edsair.doi.dedup.....708a06a05dc236486abebc8e82acb464