A Novel mutation L619F in the cardiac Na channel SCN5A associated with long-QT syndrome (LQT3): a role for the I-II linker in inactivation gating

MLA

Pieter A. Doevendans, et al. “A Novel Mutation L619F in the Cardiac Na Channel SCN5A Associated with Long-QT Syndrome (LQT3): A Role for the I-II Linker in Inactivation Gating.” Human Mutation, vol. 21, May 2003, p. 552. EBSCOhost, https://doi.org/10.1002/humu.9136.

APA

Pieter A. Doevendans, Marc A. Vos, Marc Gewillig, Robert S. Kass, Roselie Jongbloed, Hein J.J. Wellens, Xander H.T. Wehrens, Tom Rossenbacker, & Hein Heidbuchel. (2003). A Novel mutation L619F in the cardiac Na channel SCN5A associated with long-QT syndrome (LQT3): a role for the I-II linker in inactivation gating. Human Mutation, 21, 552. https://doi.org/10.1002/humu.9136

Chicago

Pieter A. Doevendans, Marc A. Vos, Marc Gewillig, Robert S. Kass, Roselie Jongbloed, Hein J.J. Wellens, Xander H.T. Wehrens, Tom Rossenbacker, and Hein Heidbuchel. 2003. “A Novel Mutation L619F in the Cardiac Na Channel SCN5A Associated with Long-QT Syndrome (LQT3): A Role for the I-II Linker in Inactivation Gating.” Human Mutation 21 (May): 552. doi:10.1002/humu.9136.