Production of truncated MBD4 protein by frameshift mutation in DNA mismatch repair-deficient cells enhances 5-fluorouracil sensitivity that is independent of hMLH1 status

MLA

Shigeru Kanaoka, et al. Production of Truncated MBD4 Protein by Frameshift Mutation in DNA Mismatch Repair-Deficient Cells Enhances 5-Fluorouracil Sensitivity That Is Independent of HMLH1 Status. Apr. 2016. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....a101c6a5cd67d12f930f9edc298577b9&authtype=sso&custid=ns315887.

APA

Shigeru Kanaoka, Satoshi Suzuki, Stephanie Tseng-Rogenski, John M. Carethers, Moriya Iwaizumi, Ken Sugimoto, Yasushi Hamaya, & Hiroaki Miyajima. (2016). Production of truncated MBD4 protein by frameshift mutation in DNA mismatch repair-deficient cells enhances 5-fluorouracil sensitivity that is independent of hMLH1 status.

Chicago

Shigeru Kanaoka, Satoshi Suzuki, Stephanie Tseng-Rogenski, John M. Carethers, Moriya Iwaizumi, Ken Sugimoto, Yasushi Hamaya, and Hiroaki Miyajima. 2016. “Production of Truncated MBD4 Protein by Frameshift Mutation in DNA Mismatch Repair-Deficient Cells Enhances 5-Fluorouracil Sensitivity That Is Independent of HMLH1 Status,” April. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....a101c6a5cd67d12f930f9edc298577b9&authtype=sso&custid=ns315887.