Cutting Edge: RIP1 Kinase Activity Is Dispensable for Normal Development but Is a Key Regulator of Inflammation in SHARPIN-Deficient Mice

MLA

Barb Swift, et al. “Cutting Edge: RIP1 Kinase Activity Is Dispensable for Normal Development but Is a Key Regulator of Inflammation in SHARPIN-Deficient Mice.” The Journal of Immunology, vol. 192, June 2014, pp. 5476–80. EBSCOhost, https://doi.org/10.4049/jimmunol.1400499.

APA

Barb Swift, Peter J. Gough, Michelle C. Schaeffer, Lauren Dare, Angela Hughes-Earle, Philip A. Harris, Carol A. Capriotti, William J. Kaiser, Edward S. Mocarski, Michael Cook, Sandy Hoffman, Viera Kasparcova, Scott B. Berger, Joshua N. Finger, & John Bertin. (2014). Cutting Edge: RIP1 Kinase Activity Is Dispensable for Normal Development but Is a Key Regulator of Inflammation in SHARPIN-Deficient Mice. The Journal of Immunology, 192, 5476–5480. https://doi.org/10.4049/jimmunol.1400499

Chicago

Barb Swift, Peter J. Gough, Michelle C. Schaeffer, Lauren Dare, Angela Hughes-Earle, Philip A. Harris, Carol A. Capriotti, et al. 2014. “Cutting Edge: RIP1 Kinase Activity Is Dispensable for Normal Development but Is a Key Regulator of Inflammation in SHARPIN-Deficient Mice.” The Journal of Immunology 192 (June): 5476–80. doi:10.4049/jimmunol.1400499.