Cite
Therapeutic Silencing of Bcl-2 by Systemically Administered siRNA Nanotherapeutics Inhibits Tumor Growth by Autophagy and Apoptosis and Enhances the Efficacy of Chemotherapy in Orthotopic Xenograft Models of ER (−) and ER (+) Breast Cancer
MLA
Ugur Akar, et al. “Therapeutic Silencing of Bcl-2 by Systemically Administered SiRNA Nanotherapeutics Inhibits Tumor Growth by Autophagy and Apoptosis and Enhances the Efficacy of Chemotherapy in Orthotopic Xenograft Models of ER (−) and ER (+) Breast Cancer.” Molecular Therapy. Nucleic Acids, vol. 2, no. 9, Sept. 2013, p. e121. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....d145c1fe4a59ff8ff66c63862a4a85de&authtype=sso&custid=ns315887.
APA
Ugur Akar, Cristian Ayugo-Rodriguez, Bulent Ozpolat, Ibrahim Tekedereli, Gabriel Lopez-Berestein, Erkan Yuca, Anil K. Sood, He Dong Han, & S. Neslihan Alpay. (2013). Therapeutic Silencing of Bcl-2 by Systemically Administered siRNA Nanotherapeutics Inhibits Tumor Growth by Autophagy and Apoptosis and Enhances the Efficacy of Chemotherapy in Orthotopic Xenograft Models of ER (−) and ER (+) Breast Cancer. Molecular Therapy. Nucleic Acids, 2(9), e121.
Chicago
Ugur Akar, Cristian Ayugo-Rodriguez, Bulent Ozpolat, Ibrahim Tekedereli, Gabriel Lopez-Berestein, Erkan Yuca, Anil K. Sood, He Dong Han, and S. Neslihan Alpay. 2013. “Therapeutic Silencing of Bcl-2 by Systemically Administered SiRNA Nanotherapeutics Inhibits Tumor Growth by Autophagy and Apoptosis and Enhances the Efficacy of Chemotherapy in Orthotopic Xenograft Models of ER (−) and ER (+) Breast Cancer.” Molecular Therapy. Nucleic Acids 2 (9): e121. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsair&AN=edsair.doi.dedup.....d145c1fe4a59ff8ff66c63862a4a85de&authtype=sso&custid=ns315887.