DBZ is a putative PPARγ agonist that prevents high fat diet-induced obesity, insulin resistance and gut dysbiosis

MLA

Pengfei Xu, et al. “DBZ Is a Putative PPARγ Agonist That Prevents High Fat Diet-Induced Obesity, Insulin Resistance and Gut Dysbiosis.” Biochimica et Biophysica Acta (BBA) - General Subjects, vol. 1861, Nov. 2017, pp. 2690–701. EBSCOhost, https://doi.org/10.1016/j.bbagen.2017.07.013.

APA

Pengfei Xu, Xiaohui Zheng, Fan Hong, Sheng Wang, Jing Wang, Tingting Xue, Jingwei Xu, Jialin Wang, Yonggong Zhai, & Xia Zhao. (2017). DBZ is a putative PPARγ agonist that prevents high fat diet-induced obesity, insulin resistance and gut dysbiosis. Biochimica et Biophysica Acta (BBA) - General Subjects, 1861, 2690–2701. https://doi.org/10.1016/j.bbagen.2017.07.013

Chicago

Pengfei Xu, Xiaohui Zheng, Fan Hong, Sheng Wang, Jing Wang, Tingting Xue, Jingwei Xu, Jialin Wang, Yonggong Zhai, and Xia Zhao. 2017. “DBZ Is a Putative PPARγ Agonist That Prevents High Fat Diet-Induced Obesity, Insulin Resistance and Gut Dysbiosis.” Biochimica et Biophysica Acta (BBA) - General Subjects 1861 (November): 2690–2701. doi:10.1016/j.bbagen.2017.07.013.