Somatic Gain of KRAS Function in the Endothelium Is Sufficient to Cause Vascular Malformations That Require MEK but Not PI3K Signaling

MLA

Meng Cui, et al. “Somatic Gain of KRAS Function in the Endothelium Is Sufficient to Cause Vascular Malformations That Require MEK but Not PI3K Signaling.” Circulation Research, vol. 127, Aug. 2020, pp. 727–43. EBSCOhost, https://doi.org/10.1161/circresaha.119.316500.

APA

Meng Cui, Emilie Boudreau, Alexander M. Herman, Dakota Gustafson, Ivan Radovanovic, Jason E. Fish, Karen Berman de Ruiz, Zhiqi Chen, Joshua D. Wythe, Taylor S Schexnayder, Peter V. DiStefano, Manuel Cantu Gutierrez, Carlos Perfecto Flores-Suarez, & Christopher S. Ward. (2020). Somatic Gain of KRAS Function in the Endothelium Is Sufficient to Cause Vascular Malformations That Require MEK but Not PI3K Signaling. Circulation Research, 127, 727–743. https://doi.org/10.1161/circresaha.119.316500

Chicago

Meng Cui, Emilie Boudreau, Alexander M. Herman, Dakota Gustafson, Ivan Radovanovic, Jason E. Fish, Karen Berman de Ruiz, et al. 2020. “Somatic Gain of KRAS Function in the Endothelium Is Sufficient to Cause Vascular Malformations That Require MEK but Not PI3K Signaling.” Circulation Research 127 (August): 727–43. doi:10.1161/circresaha.119.316500.