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[How some commensal bacteria would exacerbate colorectal carcinogenesis?]
- Source :
- médecine/sciences, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-182. ⟨10.1051/medsci/20163202011⟩, publications.edpsciences.org/fr/, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-82. ⟨10.1051/medsci/20163202011⟩, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-82. ⟨10.1051/medsci/20163202011 ⟩, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-182. 〈publications.edpsciences.org/fr/〉. 〈10.1051/medsci/20163202011〉, médecine/sciences, 2016, 32 (2), pp.175-82. ⟨10.1051/medsci/20163202011⟩, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-82. 〈10.1051/medsci/20163202011 〉
- Publication Year :
- 2016
- Publisher :
- HAL CCSD, 2016.
-
Abstract
- International audience; The gut microbiota maintains a relationship with its host with strong mutual benefits. Changes in the composition of the intestinal microbiota have been detected in colorectal cancer patients to the extent that it is now considered as a real contributing factor in this pathology. In this review, we focus on three commensal bacterial species, namely Bacteroides fragilis, Fusobacterium nucleatum, and Escherichia coli, which seem to emerge as pathogens and to contribute to colorectal carcinogenesis through their inflammatory and oncogenic properties.; Le microbiote intestinal entretient une relation mutualiste forte avec l’hôte. Depuis la mise en évidence de modifications de sa composition chez les patients atteints de cancer colorectal, le microbiote intestinal est considéré comme un facteur contribuant à part entière à cette pathologie. Nous avons focalisé notre attention dans cette revue sur trois espèces bactériennes commensales, Bacteroides fragilis, Fusobacterium nucleatum et Escherichia coli, qui semblent émerger sous les traits de pathogènes et participer au développement du cancer colorectal au travers de leurs propriétés inflammatoires et oncogéniques
- Subjects :
- 0301 basic medicine
Colorectal cancer
[SDV]Life Sciences [q-bio]
enterotoxigenic bacteroides-fragilis
[SDV.CAN]Life Sciences [q-bio]/Cancer
[ SDV.MP.BAC ] Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology
Gut flora
dna
medicine.disease_cause
General Biochemistry, Genetics and Molecular Biology
[ SDV.CAN ] Life Sciences [q-bio]/Cancer
Microbiology
03 medical and health sciences
0302 clinical medicine
[ SDV.MHEP ] Life Sciences [q-bio]/Human health and pathology
medicine
Escherichia coli
mucosa
tumorisgenesis
ComputingMilieux_MISCELLANEOUS
Gastrointestinal tract
[ SDV ] Life Sciences [q-bio]
biology
fusobacterium-nucleatum
Host (biology)
General Medicine
biology.organism_classification
medicine.disease
[SDV.MP.BAC]Life Sciences [q-bio]/Microbiology and Parasitology/Bacteriology
microenvironment
3. Good health
stomatognathic diseases
tumorigenesis
030104 developmental biology
inflammation
030220 oncology & carcinogenesis
gut
cells
Bacteroides fragilis
Fusobacterium nucleatum
Carcinogenesis
colon-cancer
[SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
Subjects
Details
- Language :
- English
- ISSN :
- 07670974 and 19585381
- Database :
- OpenAIRE
- Journal :
- médecine/sciences, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-182. ⟨10.1051/medsci/20163202011⟩, publications.edpsciences.org/fr/, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-82. ⟨10.1051/medsci/20163202011⟩, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-82. ⟨10.1051/medsci/20163202011 ⟩, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-182. 〈publications.edpsciences.org/fr/〉. 〈10.1051/medsci/20163202011〉, médecine/sciences, 2016, 32 (2), pp.175-82. ⟨10.1051/medsci/20163202011⟩, médecine/sciences, EDP Sciences, 2016, 32 (2), pp.175-82. 〈10.1051/medsci/20163202011 〉
- Accession number :
- edsair.doi.dedup.....fffea8aeba1d90f7a3adf9e04ccd2d03
- Full Text :
- https://doi.org/10.1051/medsci/20163202011⟩