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The ABCG2 Q141K hyperuricemia and gout associated variant illuminates the physiology of human urate excretion

Authors :
Kazi Mirajul Hoque
Eryn E. Dixon
Raychel M. Lewis
Jordyn Allan
Gregory D. Gamble
Amanda J. Phipps-Green
Victoria L. Halperin Kuhns
Anne M. Horne
Lisa K. Stamp
Tony R. Merriman
Nicola Dalbeth
Owen M. Woodward
Source :
Nature Communications, Vol 11, Iss 1, Pp 1-15 (2020)
Publication Year :
2020
Publisher :
Nature Portfolio, 2020.

Abstract

The common ABCG2 variant Q141K contributes to hyperuricemia and gout risk. Here, using a human interventional study and a new orthologous mouse model, the authors report a tissue specific pathobiology of the Q141K variant, and support a significant role for ABCG2 in urate excretion in both the kidney and intestine.

Subjects

Subjects :
Science

Details

Language :
English
ISSN :
20411723 and 63333244
Volume :
11
Issue :
1
Database :
Directory of Open Access Journals
Journal :
Nature Communications
Publication Type :
Academic Journal
Accession number :
edsdoj.12c73f6333324432b9f2c84850631506
Document Type :
article
Full Text :
https://doi.org/10.1038/s41467-020-16525-w