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Targeting KRAS4A splicing through the RBM39/DCAF15 pathway inhibits cancer stem cells

Authors :
Wei-Ching Chen
Minh D. To
Peter M. K. Westcott
Reyno Delrosario
Il-Jin Kim
Mark Philips
Quan Tran
Saumya R. Bollam
Hani Goodarzi
Nora Bayani
Olga Mirzoeva
Allan Balmain
Source :
Nature Communications, Vol 12, Iss 1, Pp 1-14 (2021)
Publication Year :
2021
Publisher :
Nature Portfolio, 2021.

Abstract

Kras is frequently mutated in lung cancer and two isoforms are generated via alternative splicing. Here, the authors show that the two isoforms have divergent roles in cancer stem cells and the main tumour cell population, which are regulated by hypoxia and endoplasmic reticulum stress.

Subjects

Subjects :
Science

Details

Language :
English
ISSN :
20411723
Volume :
12
Issue :
1
Database :
Directory of Open Access Journals
Journal :
Nature Communications
Publication Type :
Academic Journal
Accession number :
edsdoj.22b5d5de40945018011a5128be0357a
Document Type :
article
Full Text :
https://doi.org/10.1038/s41467-021-24498-7