Cite
Disease Phenotypes and Mechanisms of iPSC-Derived Cardiomyocytes From Brugada Syndrome Patients With a Loss-of-Function SCN5A Mutation
MLA
Wener Li, et al. “Disease Phenotypes and Mechanisms of IPSC-Derived Cardiomyocytes From Brugada Syndrome Patients With a Loss-of-Function SCN5A Mutation.” Frontiers in Cell and Developmental Biology, vol. 8, Oct. 2020. EBSCOhost, https://doi.org/10.3389/fcell.2020.592893.
APA
Wener Li, Michael Stauske, Xiaojing Luo, Stefan Wagner, Meike Vollrath, Carola S. Mehnert, Mario Schubert, Lukas Cyganek, Simin Chen, Sayed-Mohammad Hasheminasab, Gerald Wulf, Ali El-Armouche, Lars S. Maier, Gerd Hasenfuss, & Kaomei Guan. (2020). Disease Phenotypes and Mechanisms of iPSC-Derived Cardiomyocytes From Brugada Syndrome Patients With a Loss-of-Function SCN5A Mutation. Frontiers in Cell and Developmental Biology, 8. https://doi.org/10.3389/fcell.2020.592893
Chicago
Wener Li, Michael Stauske, Xiaojing Luo, Stefan Wagner, Meike Vollrath, Carola S. Mehnert, Mario Schubert, et al. 2020. “Disease Phenotypes and Mechanisms of IPSC-Derived Cardiomyocytes From Brugada Syndrome Patients With a Loss-of-Function SCN5A Mutation.” Frontiers in Cell and Developmental Biology 8 (October). doi:10.3389/fcell.2020.592893.