Targeted deletion of all isoforms of the trkC gene suggests the use of alternate receptors by its ligand neurotrophin-3 in neuronal development and implicates trkC in normal cardiogenesis

MLA

Tessarollo, Lino, et al. “Targeted Deletion of All Isoforms of the TrkC Gene Suggests the Use of Alternate Receptors by Its Ligand Neurotrophin-3 in Neuronal Development and Implicates TrkC in Normal Cardiogenesis.” Proceedings of the National Academy of Sciences of the United States, vol. 94, no. 26, Dec. 1997, p. 14776. EBSCOhost, widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsggo&AN=edsgcl.56326909&authtype=sso&custid=ns315887.

APA

Tessarollo, L., Tsoulfas, P., Donovan, M. J., Palko, M. E., Blair-Flynn, J., Hempstead, B. J., & Parada, L. F. (1997). Targeted deletion of all isoforms of the trkC gene suggests the use of alternate receptors by its ligand neurotrophin-3 in neuronal development and implicates trkC in normal cardiogenesis. Proceedings of the National Academy of Sciences of the United States, 94(26), 14776.

Chicago

Tessarollo, Lino, Pantelis Tsoulfas, Michael J. Donovan, Mary Ellen Palko, Janet Blair-Flynn, Barbara J. Hempstead, and Luis F. Parada. 1997. “Targeted Deletion of All Isoforms of the TrkC Gene Suggests the Use of Alternate Receptors by Its Ligand Neurotrophin-3 in Neuronal Development and Implicates TrkC in Normal Cardiogenesis.” Proceedings of the National Academy of Sciences of the United States 94 (26): 14776. http://widgets.ebscohost.com/prod/customlink/proxify/proxify.php?count=1&encode=0&proxy=&find_1=&replace_1=&target=https://search.ebscohost.com/login.aspx?direct=true&site=eds-live&scope=site&db=edsggo&AN=edsgcl.56326909&authtype=sso&custid=ns315887.