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1. Twenty-Four-Hour Central (Aortic) Systolic Blood Pressure: Reference Values and Dipping Patterns in Untreated Individuals

Author

Weber, Thomas, Protogerou, Athanase D, Agharazii, Mohsen, Argyris, Antonis, Aoun Bahous, Sola, Banegas, Jose R, Binder, Ronald K, Blacher, Jacques, Araujo Brandao, Andréa, Cruz, Juan J, Danninger, Kathrin, Giannatasio, Cristina, Graciani, Auxiliadora, Hametner, Bernhard, Jankowski, Piotr, Li, Yan, Maloberti, Alessandro, Mayer, Christopher C, McDonnell, Barry J, McEniery, Carmel M, Antonio Mota Gomes, Marco, Machado Gomes, Annelise, Lorenza Muiesan, Maria, Nemcsik, Janos, Paini, Anna, Rodilla, Enrique, Schutte, Aletta E, Sfikakis, Petros P, Terentes-Printzios, Dimitrios, Vallée, Alexandre, Vlachopoulos, Charalambos, Ware, Lisa, Wilkinson, Ian, Zweiker, Robert, Sharman, James E, Wassertheurer, Siegfried, International Academic 24-Hour Ambulatory Aortic Blood Pressure Consortium (I24abc.Org), Weber, Thomas [0000-0003-0617-0417], Argyris, Antonis [0000-0002-8906-6959], Banegas, Jose R [0000-0001-6152-9862], Hametner, Bernhard [0000-0003-2048-1019], Jankowski, Piotr [0000-0001-6223-8821], Maloberti, Alessandro [0000-0002-2612-6264], McDonnell, Barry J [0000-0001-5912-1278], Antonio Mota Gomes, Marco [0000-0002-6652-1533], Machado Gomes, Annelise [0000-0001-9475-3166], Lorenza Muiesan, Maria [0000-0002-4007-9441], Terentes-Printzios, Dimitrios [0000-0003-4039-8263], Vlachopoulos, Charalambos [0000-0002-9904-3558], Sharman, James E [0000-0003-2792-0811], and Apollo - University of Cambridge Repository

Subjects
Adult, Aged, 80 and over, Male, hypertension, Adolescent, Brachial Artery, blood pressure, Blood Pressure Determination, Middle Aged, Circadian Rhythm, Young Adult, Reference Values, heart rate, Humans, Arterial Pressure, Female, Aged
Abstract

Central (aortic) systolic blood pressure (cSBP) is the pressure seen by the heart, the brain, and the kidneys. If properly measured, cSBP is closer associated with hypertension-mediated organ damage and prognosis, as compared with brachial SBP (bSBP). We investigated 24-hour profiles of bSBP and cSBP, measured simultaneously using Mobilograph devices, in 2423 untreated adults (1275 women; age, 18-94 years), free from overt cardiovascular disease, aiming to develop reference values and to analyze daytime-nighttime variability. Central SBP was assessed, using brachial waveforms, calibrated with mean arterial pressure (MAP)/diastolic BP (cSBPMAP/DBPcal), or bSBP/diastolic blood pressure (cSBPSBP/DBPcal), and a validated transfer function, resulting in 144 509 valid brachial and 130 804 valid central measurements. Averaged 24-hour, daytime, and nighttime brachial BP across all individuals was 124/79, 126/81, and 116/72 mm Hg, respectively. Averaged 24-hour, daytime, and nighttime values for cSBPMAP/DBPcal were 128, 128, and 125 mm Hg and 115, 117, and 107 mm Hg for cSBPSBP/DBPcal, respectively. We pragmatically propose as upper normal limit for 24-hour cSBPMAP/DBPcal 135 mm Hg and for 24-hour cSBPSBP/DBPcal 120 mm Hg. bSBP dipping (nighttime-daytime/daytime SBP) was -10.6 % in young participants and decreased with increasing age. Central SBPSBP/DBPcal dipping was less pronounced (-8.7% in young participants). In contrast, cSBPMAP/DBPcal dipping was completely absent in the youngest age group and less pronounced in all other participants. These data may serve for comparison in various diseases and have potential implications for refining hypertension diagnosis and management. The different dipping behavior of bSBP versus cSBP requires further investigation.

Published
2022
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4. Pericarditis and pericardial effusion: one or two distinct diseases?

Author

Lazaros, George Lazarou, Emilia Vlachopoulos, Charalambos Antonopoulos, Alexios Tsioufis, Konstantinos

Abstract

The main pericardial syndromes include acute and recurrent pericarditis, constrictive pericarditis and chronic pericardial effusion in the absence of overt inflammation. Despite recent advances in pericardial syndromes, certain clinical scenarios depict remarkable peculiarities and their management is often challenging for the clinician. Acute pericarditis is the most common pericardial disease and in most instances is accompanied by pericardial effusion. On the other hand, pericardial effusion may appear as a separate clinical entity occasionally characterized by absence of inflammatory markers elevation. In cases that effusions are accompanied by C-reactive protein (CRP) elevation, the administration of empiric anti-inflammatory treatment as in acute pericarditis, is the guidelines recommended approach. Conversely, the optimal management of patients with pericardial effusions in the absence of clinical or subclinical inflammation (as depicted by CRP levels and cardiac magnetic resonance findings), is not supported by solid evidence. Patients with chronic pericardial effusions should be followed in specialized centers according to tailored timelines, based on the specific clinical scenarios which should take into account etiology, effusion size, disease duration and stability as regards symptoms and effusion volume. Patients should also be advised to seek medical care at any time if symptoms like chest pain, dyspnea and fatigue should appear.

Published
2021

5. 2017 ESC Guidelines on the Diagnosis and Treatment of Peripheral Arterial Diseases, in collaboration with the European Society for Vascular Surgery (ESVS)

Author

Aboyans, Victor, Ricco, Jean-Baptiste, Bartelink, Marie-Louise E. L., Björck, Martin, Brodmann, Marianne, Cohnert, Tina, Collet, Jean-Philippe, Czerny, Martin, De Carlo, Marco, Debus, Sebastian, Espinola-Klein, Christine, Kahan, Thomas, Kownator, Serge, Mazzolai, Lucia, Naylor, A Ross, Roffi, Marco, Röther, Joachim, Sprynger, Muriel, Tendera, Michal, Tepe, Gunnar, Venermo, Maarit, Vlachopoulos, Charalambos, Desormais, Ileana, Widimsky, Petr, Kolh, Philippe, Agewall, Stefan, Bueno, Héctor, Coca, Antonio, De Borst, Gert J., Delgado, Victoria, Dick, Florian, Erol, Cetin, Ferrini, Marc, Kakkos, Stavros, Katus, Hugo A., Knuuti, Juhani, Lindholt, Jes, Mattle, Heinrich, Pieniazek, Piotr, Piepoli, Massimo Francesco, Scheinert, Dierk, Sievert, Horst, Simpson, Iain, Sulzenko, Jakub, Tamargo, Juan, Tokgozoglu, Lale, Torbicki, Adam, Tsakountakis, Nikolaos, Tuñón, José, De Ceniga, Melina Vega, Windecker, Stephan, Zamorano, Jose Luis, Barbato, Emanuele, Coman, Ioan Mircea, Dean, Veronica, Fitzsimons, Donna, Gaemperli, Oliver, Hindricks, Gerhard, Iung, Bernard, Juni, Peter, Lancellotti, Patrizio, Leclercq, Christophe, McDonagh, Theresa, Ponikowski, Piotr, Richter, Dimitrios J., Shlyakhto, Evgeny, Simpson, Iain A., Zelveian, Parounak H., Haumer, Markus, Isachkin, Dzmitry, De Backer, Tine, Dilic, Mirza, Petrov, Ivo, Kirhmajer, Majda Vrkic, Karetova, Debora, Prescott, Eva, Soliman, Hamdy, Paapstel, Ants, Makinen, Kimmo, Tosev, Slavco, Messas, Emmanuel, Pagava, Zurab, Müller, Oliver J., Naka, Katerina K., Járai, Zoltán, Gudjonsson, Thorbjorn, Jonas, Michael, Novo, Salvatore, Ibrahimi, Pranvera, Lunegova, Olga, Dzerve, Vilnis, Misonis, Nerijus, Beissel, Jean, Pllaha, Elton, Taberkant, Mustapha, Bakken, Torbjørn, Teles, Rui, Lighezan, Daniel, Konradi, Alexandra, Zavatta, Marco, Madaric, Juraj, Fras, Zlatko, Melchor, Lorenzo Silva, Näslund, Ulf, Amann-Vesti, Beatrice, Obiekezie, Agu, Aboyans, Victor, Ricco, Jean-Baptiste, Bartelink, Marie-Louise E.L., Björck, Martin, Brodmann, Marianne, Cohnert, Tina, Collet, Jean-Philippe, Czerny, Martin, De Carlo, Marco, Debus, Sebastian, Espinola-Klein, Christine, Kahan, Thoma, Kownator, Serge, Mazzolai, Lucia, Naylor, A Ro, Roffi, Marco, Röther, Joachim, Sprynger, Muriel, Tendera, Michal, Tepe, Gunnar, Venermo, Maarit, Vlachopoulos, Charalambo, Desormais, Ileana, Widimsky, Petr, Kolh, Philippe, Agewall, Stefan, Bueno, Héctor, Coca, Antonio, De Borst, Gert J., Delgado, Victoria, Dick, Florian, Erol, Cetin, Ferrini, Marc, Kakkos, Stavro, Katus, Hugo A., Knuuti, Juhani, Lindholt, Je, Mattle, Heinrich, Pieniazek, Piotr, Piepoli, Massimo Francesco, Scheinert, Dierk, Sievert, Horst, Simpson, Iain, Sulzenko, Jakub, Tamargo, Juan, Tokgozoglu, Lale, Torbicki, Adam, Tsakountakis, Nikolao, Tuñón, José, De Ceniga, Melina Vega, Windecker, Stephan, Zamorano, Jose Lui, Barbato, Emanuele, Coman, Ioan Mircea, Dean, Veronica, Fitzsimons, Donna, Gaemperli, Oliver, Hindricks, Gerhard, Iung, Bernard, Juni, Peter, Lancellotti, Patrizio, Leclercq, Christophe, McDonagh, Theresa, Ponikowski, Piotr, Richter, Dimitrios J., Shlyakhto, Evgeny, Simpson, Iain A., Zelveian, Parounak H., Haumer, Marku, Isachkin, Dzmitry, De Backer, Tine, Dilic, Mirza, Petrov, Ivo, Kirhmajer, Majda Vrkic, Karetova, Debora, Prescott, Eva, Soliman, Hamdy, Paapstel, Ant, Makinen, Kimmo, Tosev, Slavco, Messas, Emmanuel, Pagava, Zurab, Müller, Oliver J., Naka, Katerina K., Járai, Zoltán, Gudjonsson, Thorbjorn, Jonas, Michael, Novo, Salvatore, Ibrahimi, Pranvera, Lunegova, Olga, Dzerve, Vilni, Misonis, Neriju, Beissel, Jean, Pllaha, Elton, Taberkant, Mustapha, Bakken, Torbjørn, Teles, Rui, Lighezan, Daniel, Konradi, Alexandra, Zavatta, Marco, Madaric, Juraj, Fras, Zlatko, Melchor, Lorenzo Silva, Näslund, Ulf, Amann-Vesti, Beatrice, and Obiekezie, Agu

Subjects
medicine.medical_specialty, Carotid artery disease, business.industry, Atherosclerotic disease, Upper extremity artery disease, Guideline, 030204 cardiovascular system & hematology, Vascular surgery, Lower extremity artery disease, Multisite artery disease, Peripheral Arterial Diseases, 3. Good health, 03 medical and health sciences, 0302 clinical medicine, Renal artery disease, Internal medicine, Peripheral arterial disease, medicine, Cardiology, Mesenteric artery disease, 030212 general & internal medicine, Vertebral artery disease, Cardiology and Cardiovascular Medicine, business
Abstract

2017 ESC Guidelines on the Diagnosis and Treatment of Peripheral Arterial Diseases, in collaboration with the European Society for Vascular Surgery (ESVS) : Document covering atherosclerotic disease of extracranial carotid and vertebral, mesenteric, renal, upper and lower extremity arteries

Published
2017

6. 2017 ESC Guidelines on the Diagnosis and Treatment of Peripheral Arterial Diseases, in collaboration with the European Society for Vascular Surgery (ESVS): Document covering atherosclerotic disease of extracranial carotid and vertebral, mesenteric, renal, upper and lower extremity arteriesEndorsed by: the European Stroke Organization (ESO)The Task Force for the Diagnosis and Treatment of Peripheral Arterial Diseases of the European Society of Cardiology (ESC) and of the European Society for Vascular Surgery (ESVS)

Author

Aboyans, Victor, Ricco, J. B., Bartelink, Marie-Louise E. L., Björck, Martin, Brodmann, Marianne, Cohnert, Tina, Collet, Jean-Philippe, Czerny, Martin, De Carlo, Marco, Debus, Sebastian, Espinola-Klein, Christine, Kahan, Thomas, Kownator, Serge, Mazzolai, Lucia, Naylor, A. Ross, Roffi, Marco, Röther, Joachim, Sprynger, Muriel, Tendera, Michal, Tepe, Gunnar, Venermo, Maarit, Vlachopoulos, Charalambos, Desormais, Ileana, ESC Scientific Document Group, Verisuonikirurgian yksikkö, Department of Surgery, Clinicum, and HUS Abdominal Center

Subjects
Peripheral Arterial Disease, Fibrinolytic Agents, Risk Factors, 3121 General medicine, internal medicine and other clinical medicine, Humans, Vascular Surgical Procedures
Abstract

Endorsed by: the European Stroke Organization (ESO), The Task Force for the Diagnosis and Treatment of Peripheral Arterial Diseases of the European Society of Cardiology (ESC) and of the European Society for Vascular Surgery (ESVS)

Published
2017

8. Wytyczne ESC/EAS dotyczące leczenia zaburzeń lipidowych w 2016 roku

Author

Catapano, Alberico L, Graham, Ian, De Backer, Guy, Wiklund, Olov, Chapman, M John, Drexel, Heinz, Hoes, Arno W, Jennings, Catriona S, Landmesser, Ulf, Pedersen, Terje R, Reiner, Željko, Riccardi, Gabriele, Taskinen, Marja-Riita, Tokgozoglu, Lale, Verschuren, W M Monique, Vlachopoulos, Charalambos, Wood, David A, and Zamorano, Jose Luis

Subjects
apolipoproteina B, remnanty lipoprotein, całkowite ryzyko sercowo-naczyniowe, Practice Guideline, zaburzenie lipidowe, cholesterol, triglicerydy, lipoproteiny o małej gęstości, lipoproteiny o dużej gęstości, Journal Article
Published
2016

12. Vascular consequences of inflammation: a position statement from the ESH Working Group on Vascular Structure and Function and the ARTERY Society

Author

Maki-Petaja, Kaisa, Zanoli, Luca, Briet, Marie, Empana, Jean Phillippe, Guimarães, Cunha Pedro, Protegerou, Athanase D, Tedgui, Alain, Touyz, Rhian M, Schiffrin, Ernesto L, Spronck, Bart, Bouchard, Philippe, Vlachopoulos, Charalambos, Bruno, Rosa Maria, and Boutouyrie, Pierre

Subjects
Arthritis, Rheumatoid, Inflammation, Vascular Stiffness, Heart Disease Risk Factors, Humans, Arteries, Vascular Diseases, Atherosclerosis, Inflammatory Bowel Diseases, 3. Good health
Abstract

Inflammation is a physiological response to aggression of pathogenic agents aimed at eliminating the aggressor agent and promoting healing. Excessive inflammation, however, may contribute to tissue damage and an alteration of arterial structure and function. Increased arterial stiffness is a well-recognized cardiovascular risk factor independent of blood pressure levels and an intermediate endpoint for cardiovascular events. In the present review we discuss immune mediated mechanisms by which inflammation can influence arterial physiology and lead to vascular dysfunction such as atherosclerosis and arterial stiffening. We also show that acute inflammation predisposes the vasculature to arterial dysfunction and stiffening, and alteration of endothelial function and that chronic inflammatory diseases such as rheumatoid arthritis, inflammatory bowel disease and psoriasis are accompanied by profound arterial dysfunction which is proportional to the severity of inflammation. Current findings suggest that treatment of inflammation by targeted drugs leads to regression of arterial dysfunction. There is hope that these treatments will improve outcomes for patients.

13. Central aortic pressure calibration: 'a bridge too far'? Reply

Author

Papaioannou, Theodore G., Karageorgopoulou, Theofani D., Sergentanis, Theodoros N., Protogerou, Athanase D., Psaltopoulou, Theodora, Sharman, James E., Weber, Thomas, Blacher, Jacques, Daskalopoulou, Stella S., Wassertheurer, Siegfried, Khir, Ashraf W., Vlachopoulos, Charalambos, Stergiopulos, Nikolaos, Stefanadis, Christodoulos, Nichols, Wilmer W., and Tousoulis, Dimitrios

15. Validation of non-invasive central blood pressure devices: ARTERY Society task force consensus statement on protocol standardization

Author

Sharman, James E, Avolio, Alberto P, Baulmann, Johannes, Benetos, Athanase, Blacher, Jacques, Blizzard, C Leigh, Boutouyrie, Pierre, Chen, Chen-Huan, Chowienczyk, Phil, Cockcroft, John R, Cruickshank, J Kennedy, Ferreira, Isabel, Ghiadoni, Lorenzo, Hughes, Alun, Jankowski, Piotr, Laurent, Stephane, McDonnell, Barry J, McEniery, Carmel, Millasseau, Sandrine C, Papaioannou, Theodoros G, Parati, Gianfranco, Park, Jeong Bae, Protogerou, Athanase D, Roman, Mary J, Schillaci, Giuseppe, Segers, Patrick, Stergiou, George S, Tomiyama, Hirofumi, Townsend, Raymond R, Van Bortel, Luc M, Wang, Jiguang, Wassertheurer, Siegfried, Weber, Thomas, Wilkinson, Ian B, and Vlachopoulos, Charalambos

Subjects
Consensus, Hypertension, Cardiology, Humans, Blood Pressure, Blood Pressure Determination, Congresses as Topic, Reference Standards, Societies, Medical, 3. Good health, Netherlands

16. A Systematic Review of the Association Between Erectile Dysfunction and Cardiovascular Disease

Author

Francesco Montorsi, Alberto Briganti, Piero Montorsi, Giorgio Gandaglia, Robert A. Kloner, Graham Jackson, Charalambos Vlachopoulos, Gandaglia, Giorgio, Briganti, Alberto, Jackson, Graham, Kloner, Robert A., Montorsi, Francesco, Montorsi, Piero, and Vlachopoulos, Charalambos

Subjects
Male, medicine.medical_specialty, Pathology, Arterie, Urology, Context (language use), Penile artery, Coronary Artery Disease, Disease, Nitric Oxide, Coronary artery disease, Coronary circulation, Erectile Dysfunction, Phosphodiesterase 5 Inhibitor, Internal medicine, Medicine, Testosterone, Endothelium, Endothelial dysfunction, Inflammation, business.industry, Artery-size hypothesi, Biomarker, Cardiovascular disease, medicine.disease, Coronary arteries, medicine.anatomical_structure, Erectile dysfunction, Phosphodiesterase type 5 inhibitor, Atherosclerosi, Screening, Cardiology, business, Human
Abstract

Context Erectile dysfunction (ED) is considered a vascular impairment that shares many risk factors with cardiovascular disease (CVD). A correlation between ED and CVD has been hypothesized, and ED has been proposed as an early marker of symptomatic CVD. Objective To analyze the relationship between ED and CVD, evaluating the pathophysiologic links between these conditions, and to identify which patients would benefit from cardiologic assessment when presenting with ED. Evidence acquisition A systematic literature review searching Medline, Embase, and Web of Science databases was performed. The search strategy included the terms erectile dysfunction, cardiovascular disease, coronary artery disease, risk factors, pathophysiology, atherosclerosis, low androgen levels, inflammation, screening, and phosphodiesterase type 5 inhibitors alone or in combination. We limited our search to studies published between January 2005 and May 2013. Evidence synthesis Several studies reported an association between ED and CVD. The link between these conditions might reside in the interaction between androgens, chronic inflammation, and cardiovascular risk factors that determines endothelial dysfunction and atherosclerosis, resulting in disorders of penile and coronary circulation. Because penile artery size is smaller compared with coronary arteries, the same level of endothelial dysfunction causes a more significant reduction of blood flow in erectile tissues compared with that in coronary circulation. Thus ED could be an indicator of systemic endothelial dysfunction. From a clinical standpoint, because ED may precede CVD, it can be used as an early marker to identify men at higher risk of CVD events. ED patients at high risk of CVD should undergo detailed cardiologic assessment and receive intensive treatment of risk factors. Conclusions ED and CVD should be regarded as two different manifestations of the same systemic disorder. ED usually precedes CVD onset, and it might be considered an early marker of symptomatic CVD. © 2013 European Association of Urology.

Published
2014

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