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38 results on '"Anil K. Challa"'

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1. Engaging students in a genetics course-based undergraduate research experience utilizing Caenorhabditis elegans in hybrid learning to explore human disease gene variants

2. Identification of Nrf2-responsive microRNA networks as putative mediators of myocardial reductive stress

3. Loss of Brain Angiogenesis Inhibitor-3 (BAI3) G-Protein Coupled Receptor in Mice Regulates Adaptive Thermogenesis by Enhancing Energy Expenditure

4. Validation of gene editing efficiency with CRISPR-Cas9 system directly in rat zygotes using electroporation mediated delivery and embryo culture

5. Teleological role of L-2-hydroxyglutarate dehydrogenase in the kidney

6. Exercise Mediated Nrf2 Signaling Protects the Myocardium From Isoproterenol-Induced Pathological Remodeling

7. Integrating CRISPR-Cas9 Technology into Undergraduate Courses: Perspectives from a National Science Foundation (NSF) Workshop for Undergraduate Faculty, June 2018

8. Increased trabecular bone and improved biomechanics in an osteocalcin-null rat model created by CRISPR/Cas9 technology

9. Zebrafish Tumor Graft Transplantation to Grow Tumors In Vivo That Engraft Poorly as Single Cell Suspensions

10. Identification of Nrf2-responsive microRNA networks as putative mediators of myocardial reductive stress

13. Transcriptional Regulation of Structural and Functional Adaptations in a Developing Adulthood Myocardium

14. Heterozygous Pkhd1C642* mice develop cystic liver disease and proximal tubule ectasia that mimics radiographic signs of medullary sponge kidney

15. A mixing heteroduplex mobility assay (mHMA) to genotype homozygous mutants with small indels generated by CRISPR-Cas9 nucleases

16. Zebrafish Tumor Graft Transplantation to Grow Tumors

17. Student Perceptions of Authoring a Publication Stemming from a Course-Based Undergraduate Research Experience (CURE)

18. Physiological and metabolic characteristics of novel double-mutant female mice with targeted disruption of both growth hormone-releasing hormone and growth hormone receptor

19. Transcriptional regulation of structural and functional adaptations in a developing adulthood myocardium

20. Teleological role of L-2-hydroxyglutarate dehydrogenase in the kidney

21. Physiological and Metabolic Features of Mice with CRISPR/Cas9-Mediated Loss-of-Function in Growth Hormone-Releasing Hormone

22. Generation of a GLO-2 deficient mouse reveals its effects on liver carbonyl and glutathione levels

23. Integrating CRISPR-Cas9 Technology into Undergraduate Courses: Perspectives from a National Science Foundation (NSF) Workshop for Undergraduate Faculty, June 2018

25. ZEBRAFISH AS MODELS OF TYROSINE KINASE‐DRIVEN CANCER

26. Truncating PKHD1 and PKD2 mutations alter energy metabolism

27. Analysis of novel domain-specific mutations in the zebrafish

28. NF1 deficiency correlates with estrogen receptor signaling and diminished survival in breast cancer

29. Analysis of novel domain-specific mutations in the zebrafishndr2/cyclopsgene generated using CRISPR-Cas9 RNPs

30. Cardiac specific Nrf2 expression protects the myocardium from isoproterenol-induced pathological remodeling

31. Enhanced Keap1-Nrf2 signaling protects the myocardium from isoproterenol-induced pathological remodeling in mice

32. Mutation of Growth Arrest Specific 8 Reveals a Role in Motile Cilia Function and Human Disease

33. Novel Hypomorphic Alleles of the Mouse Tyrosinase Gene Induced by CRISPR-Cas9 Nucleases Cause Non-Albino Pigmentation Phenotypes

34. Abstract B43: NF1 deficiency induces aggressive mammary carcinomas in a CRISPR rat model and correlates with poor outcome in sporadic human breast cancer

35. The complete mitochondrial genome of Asian palm civet (Paradoxurus hermaphroditus) with phylogenetic consideration

36. Defining function of wild-type and three patient-specific TP53 mutations in a zebrafish model of embryonal rhabdomyosarcoma

37. Mutation of Growth Arrest Specific 8 Reveals a Role in Motile Cilia Function and Human Disease.

38. Novel Hypomorphic Alleles of the Mouse Tyrosinase Gene Induced by CRISPR-Cas9 Nucleases Cause Non-Albino Pigmentation Phenotypes.

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