195 results on '"Obeng, Esther A."'
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2. Author Correction: Proteasome inhibition targets the KMT2A transcriptional complex in acute lymphoblastic leukemia
3. Venetoclax-based therapy as a bridge to allogeneic hematopoietic cell transplantation in children with relapsed/refractory AML
4. An inflammatory state remodels the immune microenvironment and improves risk stratification in acute myeloid leukemia
5. Murine foetal liver supports limited detectable expansion of life-long haematopoietic progenitors
6. SF3B1 mutation and ATM deletion codrive leukemogenesis via centromeric R-loop dysregulation
7. Engineering naturally occurring CD7− T cells for the immunotherapy of hematological malignancies
8. Splicing factor deficits render hematopoietic stem and progenitor cells sensitive to STAT3 inhibition
9. Preferential expansion of CD8+ CD19-CAR T cells postinfusion and the role of disease burden on outcome in pediatric B-ALL
10. A Murine Model of Chronic Lymphocytic Leukemia Based on B Cell-Restricted Expression of Sf3b1 Mutation and Atm Deletion
11. Murine fetal bone marrow does not support functional hematopoietic stem and progenitor cells until birth
12. Author Correction: An inflammatory state remodels the immune microenvironment and improves risk stratification in acute myeloid leukemia
13. Memory T-cell enriched haploidentical transplantation with NK cell addback results in promising long-term outcomes: a phase II trial.
14. Clones assemble! The clonal complexity of blood during ontogeny and disease
15. Acute Kidney Injury in Children: A 3-Year Retrospective Analysis at Komfo Anokye Teaching Hospital, Ghana
16. The development of therapy related myeloid neoplasms in childhood cancer survivors
17. SF3B1 mutation and ATM deletion co-drive leukemogenesis via centromeric R-loop dysregulation
18. Congenital macrothrombocytopenia with focal myelofibrosis due to mutations in human G6b-B is rescued in humanized mice
19. Therapeutic approaches targeting splicing factor mutations in myelodysplastic syndromes and acute myeloid leukemia
20. Supplementary Excel Tables from Integrative Genomic Analysis of Pediatric Myeloid-Related Acute Leukemias Identifies Novel Subtypes and Prognostic Indicators
21. Supplementary Tables and Figures from Integrative Genomic Analysis of Pediatric Myeloid-Related Acute Leukemias Identifies Novel Subtypes and Prognostic Indicators
22. Data from Integrative Genomic Analysis of Pediatric Myeloid-Related Acute Leukemias Identifies Novel Subtypes and Prognostic Indicators
23. Supplementary table 1 from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations
24. Supplementary table 3 from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations
25. Supplementary figures and legends from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations
26. Supplementary methods from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations
27. Supplementary table 2 from Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations
28. 3211 – SF3B1/DNMT3A CO-MUTATIONS MITIGATES DNMT3A PHENOTYPE IN HEMATOPOIETIC STEM AND PROGENITOR CELLS
29. 3138 – EPIGENETIC REGULATORS TET2 AND POLYCOMB REPRESSIVE COMPLEX 2 (PRC2) COORDINATE GENE EXPRESSION IN MYELODYSPLASTIC SYNDROME (MDS)
30. An inflammatory state remodels the immune microenvironment and improves risk stratification in acute myeloid leukemia
31. Venetoclax-based therapy as a bridge to allogeneic hematopoietic cell transplantation in children with relapsed/refractory AML
32. A Murine Model Harboring Cooperating DNMT3A and SF3B1 Mutations Phenocopies SF3B1 Driven Myelodysplastic Syndrome
33. Mutant SF3B1 splices a more leukemogenic EVI1
34. CD45RA Depleted T-Cell Addback and Prophylactic Blinatumomab Administration Following Tcrαβ/CD19-Depleted Haploidentical Transplantation in Pediatric Patients with High Risk Acute Leukemia
35. CD45RA-Depleted Haploidentical Transplantation Combined with NK Cell Addback Results in Promising Long-Term Outcomes in Pediatric Patients with High-Risk Hematologic Malignancies
36. Venetoclax-Based Combination Therapy As a Bridge to Allogeneic Hematopoietic Stem Cell Transplant in Children with Relapsed/Refractory AML
37. Towards SDG3: Empowering Non-Nephrologists to Perform Peritoneal Dialysis in District Settings
38. Demography and Outcomes of End-Stage Renal Diseases (ESRD) of in Children Admitted to a Renal Unit in a Teaching Hospital in Kumasi, Ghana.
39. 3220 – SF3B1/DNMT3A CO-MUTATION MIMICS FEATURES OF SOLE SF3B1 OR DNMT3A MUTATION DEPENDING ON STRESS CONDITIONS
40. 3137 – A MODIFIED ACTIVIN RECEPTOR TYPE IIB LIGAND TRAP (RAP-536) IMPROVES ANEMIA IN SF3B1K700E MICE BY PROMOTING EARLY-STAGE ERYTHROPOIESIS AND IMPROVING ALTERNATIVE SPLICING
41. CD45RA-Depleted Haploidentical Transplantation Combined with NK Cell Addback Results in Promising Long-Term Outcomes in Pediatric Patients with High-Risk Hematologic Malignancies
42. CD45RO+ T-Cell Add Back and Prophylactic Blinatumomab Administration Post Tcrαβ/CD19-Depleted Haploidentical Transplantation in Pediatric Patients with High Risk Acute Leukemia
43. Selective Targeting of Splicing Factor Mutant Hematopoietic Stem and Progenitor Cells Via STAT3 Inhibition
44. A Prospective Cohort Quality Improvement Study to Reduce the Time to Antibiotics for New Fever in Neutropenic Pediatric Oncology Inpatients
45. Integrative Genomic Analysis of Pediatric Myeloid-Related Acute Leukemias Identifies Novel Subtypes and Prognostic Indicators
46. 3116 – SELECTIVE TARGETING OF SPLICING FACTOR MUTANT HEMATOPOIETIC STEM AND PROGENITOR CELLS VIA STAT3 INHIBITION
47. Nonsense-Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells Harboring SF3B1 or U2AF1 Mutations
48. Abstract 2093: Alternative splicing in hematopoietic stem cells is affected by the loss of DNMT3A
49. Engineering naturally occurring CD7−T cells for the immunotherapy of hematological malignancies
50. Nonsense Mediated RNA Decay Is a Unique Vulnerability of Cancer Cells with SF3B1 and U2AF1 Mutations
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