Search

The Dexing porphyry copper ore mine is located in the Qin-Hang metallogenic belt between the Yangtze block and the Cathaysia block. It is a giant porphyry copper mine in China, including 3 ore districts: Tongchang, Fujiawu and Zhushahong. Our analyses of Re in molybdenite indicate that the ore-forming material of the copper ore deposits in Dexing should be mainly mantle-derived. Our study fills in a gap in the study of formation time of the Dexing copper mine, and further proves that the copper ore deposits in the three ore districts should be formed simultaneously, about 170 Ma, belonging to the early Yanshan period, and that the formation time of the copper ore deposits should be consistent with the formation time of granodiorite porphyry in which the copper ore deposits are hosted. Promising areas for seeking porphyry copper ore deposits is predicated to be the west or southwest of Dexing. [ABSTRACT FROM AUTHOR]

White pottery is among the most significant finds from China's earliest state, Erlitou ( c. 1900–1500bc). Samples were primarily discovered in small numbers from elite tombs of a few sites, leading to the hypothesis that they were made at only a few locations and then circulated regionally as prestige items. To facilitate determining provenances, we compare the ICP–MS trace elements and TIMS Sr isotopes of whiteware with two soil samples from Nanwa, a possible manufacturing site, and with shards found at three other sites: Erlitou, Huizui and Nanzhai. The Nanwa shards demonstrate special elemental and Sr isotopic features. Considering the chemical observation and archaeological background together, we propose that Nanwa was a centre for whiteware production, although the two soil samples we collected there were probably not the exact materials used. Some whiteware pieces from Erlitou, Huizui and Nanzhai fall in the chemical field defined by Nanwa samples, indicating that they were possibly made at Nanwa. Many other samples from these three sites plot outside the Nanwa field, implying they were probably not Nanwa products. This study demonstrates that while chemical sourcing is very useful, firm archaeological context must remain the cornerstone of such research. [ABSTRACT FROM AUTHOR]

The article presents the study conducted regarding the role of progesterone receptor (PGR) gene in endometrial cancer risk development in China. The study which collected DNA samples and information from Shanghai Endometrial Cancer Study aims to determine the association of PGR with the cancer risk in the Chinese population. The result shows that the gene is associated with the risk due to the polymorphism in the PGR region.

Background: Obesity is a major risk factor for endometrial cancer. Obesity, particularly central obesity, is considered as a systemic inflammatory condition and is related strongly to insulin resistance. C-reactive protein (CRP) is the most recognized biologic marker of chronic systematic inflammation, and it is conceivable that the CRP gene may work together with obesity in the development of endometrial cancer.Methods: On the basis of a population-based case-control study in a Chinese population, the authors obtained obesity measurements and data on 6 CRP single-nucleotide polymorphisms (SNPs) from 1046 patients with newly diagnosed endometrial cancer (cases) and from 1035 age frequency-matched controls. The association of the CRP SNPs with endometrial cancer risk and their modification on the association between obesity and endometrial cancer risk were evaluated.Results: Although CRP SNPs alone were not associated with endometrial cancer, the associations of endometrial cancer with central obesity, measured as the waist-to-hip ratio (WHR) and the waist circumference, seemed to be stronger in women who were homozygous for the major allele of reference SNP (rs)1130864 (cytidine [C]/C) than in women who had the C/thymidine (T) and T/T genotypes (interaction test: P = .013 for WHR; P = .083 for waist circumference). When the women were stratified further by menopausal status, the observed interactions persisted mainly in premenopausal women (interaction test: P < .001 for WHR; P = .002 for waist circumference).Conclusions: The current results suggested that, in the Chinese population that was studied, obesity-related insulin resistance and proinflammatory effects may play an important role in endometrial cancer risk, and these effects were modified significantly by the CRP SNP rs1130864. [ABSTRACT FROM AUTHOR]

The article discusses a study which analyzed the age-adjusted prevalence of general and centralized obesity among Chinese men in Shanghai. Measurement of moderate and severe centralized obesity was performed using waist-to-hip ratio. It was discovered that the prevalence of overweight was 48.8% and obesity was 10.5% among Chinese men. Higher prevalence of obesity was found in men with high income or were retired, tea drinkers or nonusers of ginseng.

The purpose of our study was to evaluate the association of menstrual and reproductive factors with the risk of endometrial cancer. In a population-based case-control study conducted in urban Shanghai, in-person interviews were completed for 833 women aged 30–69 years and an equal number of controls frequency-matched to cases by age. All cases were newly diagnosed with endometrial cancer between January 1, 1997 and December 31, 2001. The unconditional logistic regression model was employed to derive the adjusted odds ratios (ORs) of endometrial cancer and 95% confidence intervals (CIs) in relation to menstrual and reproductive factors. Earlier menarche age, particularly among premenopausal women, and later menopausal age were associated with an elevated risk of endometrial cancer. A clear dose-response relation between endometrial cancer risk and years of menstruation was observed (p for trend < 0.01). Compared to women ever having a pregnancy and women ever having had a live birth, respectively, nulligravity and nulliparity were both associated with a more than one-fold elevated risk of endometrial cancer. Both completed (OR = 3.02, 95% CI 1.10–8.32 for women never having a complete pregnancy) and incomplete pregnancy (OR = 0.69, 95%CI 0.55–0.87) conferred a protective effect against endometrial cancer, and the protective effect appeared to increase with total number of pregnancies (p for trend = 0.01). The effect of pregnancy on endometrial cancer remained unchanged with increasing time since the last pregnancy. Stillbirth and age at first pregnancy was unrelated to endometrial cancer risk. Our study suggests that prolonged menstruation was related to an increased risk of endometrial cancer while pregnancy, including induced abortion, reduced the risk of endometrial cancer. © 2003 Wiley-Liss, Inc. [ABSTRACT FROM AUTHOR]