243 results on '"Rayes, Julie"'
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2. Hot under the clot: venous thrombogenesis is an inflammatory process
3. Efficacy of platelet-inspired hemostatic nanoparticles on bleeding in von Willebrand disease murine models
4. Novel mechanisms of thrombo-inflammation during infection: spotlight on neutrophil extracellular trap-mediated platelet activation
5. S100A8/A9 drives the formation of procoagulant platelets through GPIbα
6. Basic Mechanisms of Hemolysis-Associated Thrombo-Inflammation and Immune Dysregulation
7. Illustrated State‐of‐the‐Art Capsules of the ISTH 2022 Congress
8. Platelet‐inspired nanomedicine in hemostasis thrombosis and thromboinflammation
9. Galectin‐9 activates platelet ITAM receptors glycoprotein VI and C‐type lectin‐like receptor‐2
10. Spleen tyrosine kinase inhibition mitigates hemin-induced thromboinflammation in the lung and kidney of sickle cell mice
11. The dual role of platelet‐innate immune cell interactions in thrombo‐inflammation
12. High factor VIII concentrations interfere with glycoprotein VI-mediated platelet activation in vitro
13. Discrete and conserved inflammatory signatures drive thrombosis in different organs afterSalmonellainfection
14. Hydroxychloroquine inhibits hemolysis-induced arterial thrombosis ex vivoand improves lung perfusion in hemin-treated mice
15. Modelling the pathology and treatment of cardiac fibrosis in vascularised atrial and ventricular cardiac microtissues
16. Hydroxychloroquine inhibits hemolysis-induced arterial thrombosis ex vivo and improves lung perfusion in hemin-treated mice
17. The Role of CLEC-2 in and Beyond the Vasculature
18. Functional significance of the platelet immune receptors GPVI and CLEC-2
19. Megakaryocyte NLRP3 hyperactivation induces mild anemia and potentiates inflammatory response in mice
20. Contributors
21. GPVI and CLEC-2
22. Data from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
23. Supp Table 1 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
24. Supp Table 2 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
25. Supp Table 3 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
26. Supp Table 4 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
27. Supp Table 5 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
28. Supplementary Information from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
29. Megakaryocyte NLRP3 hyperactivation induces anemia and potentiates inflammatory response in mice
30. Heme: Modulator of Plasma Systems in Hemolytic Diseases
31. Human Bone Marrow Organoids Enable the Study of Hematopoietic Cell-Stromal Interactions and Support the Survival of Malignant Cells from Patients
32. Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies
33. Severity of SARS-CoV-2 infection is associated with high numbers of alveolar mast cells and their degranulation
34. CLEC-2 Supports Platelet Aggregation in Mouse but not Human Blood at Arterial Shear
35. SARS-CoV-2 Spike- and Nucleoprotein-Specific Antibodies Induced After Vaccination or Infection Promote Classical Complement Activation
36. Preferential uptake of SARS-CoV-2 by pericytes potentiates vascular damage and permeability in an organoid model of the microvasculature
37. Mutations in the A3 domain of Von Willebrand factor inducing combined qualitative and quantitative defects in the protein
38. Complement Factor H Inhibits CD47-Mediated Resolution of Inflammation
39. It takes guts to boost platelet reactivity and inflammation
40. Adverse Outcome in COVID-19 Is Associated With an Aggravating Hypo-Responsive Platelet Phenotype
41. Platelets and Antiplatelet Medication in COVID-19-Related Thrombotic Complications
42. Mutation and ADAMTS13-dependent modulation of disease severity in a mouse model for von Willebrand disease type 2B
43. In vitro, classical complement activation differs by disease severity and between SARS-CoV-2 antigens
44. CLEC-2 Prevents Accumulation and Retention of Inflammatory Macrophages During Murine Peritonitis
45. Stimulation of vascular organoids with SARS-CoV-2 antigens increases endothelial permeability and regulates vasculopathy
46. The podoplanin-CLEC-2 axis inhibits inflammation in sepsis
47. Platelets: bridging thrombosis and inflammation
48. CLEC-2 promotes inflammatory peritoneal macrophage emigration to draining lymph nodes during endotoxemia
49. Post-translational polymodification of β1-tubulin regulates motor protein localization in platelet production and function
50. Salmonella-induced thrombi in mice develop asynchronously in the spleen and liver and are not effective bacterial traps
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