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5. S100A8/A9 drives the formation of procoagulant platelets through GPIbα

7. Illustrated State‐of‐the‐Art Capsules of the ISTH 2022 Congress

10. Spleen tyrosine kinase inhibition mitigates hemin-induced thromboinflammation in the lung and kidney of sickle cell mice

12. High factor VIII concentrations interfere with glycoprotein VI-mediated platelet activation in vitro

13. Discrete and conserved inflammatory signatures drive thrombosis in different organs afterSalmonellainfection

14. Hydroxychloroquine inhibits hemolysis-induced arterial thrombosis ex vivoand improves lung perfusion in hemin-treated mice

15. Modelling the pathology and treatment of cardiac fibrosis in vascularised atrial and ventricular cardiac microtissues

18. Functional significance of the platelet immune receptors GPVI and CLEC-2

20. Contributors

21. GPVI and CLEC-2

22. Data from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

23. Supp Table 1 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

24. Supp Table 2 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

25. Supp Table 3 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

26. Supp Table 4 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

27. Supp Table 5 from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

28. Supplementary Information from Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

31. Human Bone Marrow Organoids Enable the Study of Hematopoietic Cell-Stromal Interactions and Support the Survival of Malignant Cells from Patients

32. Human Bone Marrow Organoids for Disease Modeling, Discovery, and Validation of Therapeutic Targets in Hematologic Malignancies

33. Severity of SARS-CoV-2 infection is associated with high numbers of alveolar mast cells and their degranulation

35. SARS-CoV-2 Spike- and Nucleoprotein-Specific Antibodies Induced After Vaccination or Infection Promote Classical Complement Activation

36. Preferential uptake of SARS-CoV-2 by pericytes potentiates vascular damage and permeability in an organoid model of the microvasculature

38. Complement Factor H Inhibits CD47-Mediated Resolution of Inflammation

40. Adverse Outcome in COVID-19 Is Associated With an Aggravating Hypo-Responsive Platelet Phenotype

41. Platelets and Antiplatelet Medication in COVID-19-Related Thrombotic Complications

43. In vitro, classical complement activation differs by disease severity and between SARS-CoV-2 antigens

44. CLEC-2 Prevents Accumulation and Retention of Inflammatory Macrophages During Murine Peritonitis

45. Stimulation of vascular organoids with SARS-CoV-2 antigens increases endothelial permeability and regulates vasculopathy

46. The podoplanin-CLEC-2 axis inhibits inflammation in sepsis

48. CLEC-2 promotes inflammatory peritoneal macrophage emigration to draining lymph nodes during endotoxemia

49. Post-translational polymodification of β1-tubulin regulates motor protein localization in platelet production and function

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