151. A20-mediated deubiquitination of ERα in the microenvironment of CD163
- Author
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Qiaoying, Lv, Liying, Xie, Yali, Cheng, Yue, Shi, Weiwei, Shan, Chengcheng, Ning, Bingying, Xie, Bingyi, Yang, Xuezhen, Luo, Qizhi, He, Qin, Zhu, Yingli, Zhang, Zhenbo, Zhang, Chenji, Wang, Xiaojun, Chen, and Congjian, Xu
- Subjects
Dose-Response Relationship, Drug ,Estradiol ,Protein Stability ,Macrophages ,Estrogen Receptor alpha ,Ubiquitination ,Antigens, Differentiation, Myelomonocytic ,Apoptosis ,Receptors, Cell Surface ,Cell Communication ,Endometrial Neoplasms ,Mice, Inbred C57BL ,HEK293 Cells ,Phenotype ,Antigens, CD ,Cell Line, Tumor ,Tumor Microenvironment ,Animals ,Cytokines ,Humans ,Female ,Tumor Necrosis Factor alpha-Induced Protein 3 ,Cell Proliferation ,Half-Life ,Signal Transduction - Abstract
Continuous estrogen signaling is thought to be the main mechanism causing endometrial cancer (EC). Studies have demonstrated that CD163
- Published
- 2018