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2. Allelic regulation of Keratin 19 gene expression during pancreatic development and carcinogenesis

3. DNA methylation‐associated allelic inactivation regulates Keratin 19 gene expression during pancreatic development and carcinogenesis

4. BTK isoforms p80 and p65 are expressed in head and neck squamous cell carcinoma (HNSCC) and involved in tumor progression

6. Figure S5 Tumour growth upon ATRi and Gemcitabine Treatment in Allografts model from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

7. Figure S2 from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

8. Figure S1 GSEA Analysis from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

9. Supplementary Data from RINT1 Regulates SUMOylation and the DNA Damage Response to Preserve Cellular Homeostasis in Pancreatic Cancer

10. Table S1 from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

11. Data from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

12. Data from RINT1 Regulates SUMOylation and the DNA Damage Response to Preserve Cellular Homeostasis in Pancreatic Cancer

13. Figure S3 Tumour growth upon Olaparib and Gemcitabine Treatment in Allografts model from ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

14. Supplementary Figure S6 from HSP90 Supports Tumor Growth and Angiogenesis through PRKD2 Protein Stabilization

15. Supplementary Figure Legend from HSP90 Supports Tumor Growth and Angiogenesis through PRKD2 Protein Stabilization

16. Supplementary Methods from HSP90 Supports Tumor Growth and Angiogenesis through PRKD2 Protein Stabilization

18. BTK Isoforms p80 and p65 Are Expressed in Head and Neck Squamous Cell Carcinoma (HNSCC) and Involved in Tumor Progression

20. Organoids at the PUB: The Porcine Urinary Bladder Serves as a Pancreatic Niche for Advanced Cancer Modeling

21. Functional Genomic Screening in Human Pluripotent Stem Cells Reveals New Roadblocks in Early Pancreatic Endoderm Formation

22. CDKN2A-Mutated Pancreatic Ductal Organoids from Induced Pluripotent Stem Cells to Model a Cancer Predisposition Syndrome

23. Human Pluripotent Stem Cells Go Diabetic: A Glimpse on Monogenic Variants

24. Enteropathogenic Infections: Organoids Go Bacterial

25. Functional genomic screening during somatic cell reprogramming identifies DKK3 as a roadblock of organ regeneration

26. Telomerase and Pluripotency Factors Jointly Regulate Stemness in Pancreatic Cancer Stem Cells

27. Modeling plasticity and dysplasia of pancreatic ductal organoids derived from human pluripotent stem cells

28. Functional Genomic Screening During Somatic Cell Reprogramming Identifies DKK3 as a Roadblock of Organ Regeneration

29. Protein kinase D2 is a crucial regulator of tumour cell-endothelial cell communication in gastrointestinal tumours

30. RINT1 Regulates SUMOylation and the DNA Damage Response to Preserve Cellular Homeostasis in Pancreatic Cancer

31. Synergistic targeting and resistance to PARP inhibition in DNA damage repair-deficient pancreatic cancer

32. A feedback-loop between telomerase activity and stemness factors regulates PDAC stem cells

33. Synergistic targeting and resistance to PARP inhibition in DNA damage repair-deficient pancreatic cancer

36. Adenosine receptor 2B activity promotes autonomous growth, migration as well as vascularization of head and neck squamous cell carcinoma cells

39. Abstract 3437: Requirement for YAP1 signaling in myxoid liposarcoma

40. Variant classification in precision oncology

41. Requirement for YAP1 signaling in myxoid liposarcoma

42. Thirty-eight-negative kinase 1 mediates trauma-induced intestinal injury and multi-organ failure

44. ATM Deficiency Generating Genomic Instability Sensitizes Pancreatic Ductal Adenocarcinoma Cells to Therapy-Induced DNA Damage

46. Synergistic targeting and resistance to PARP inhibition in DNA damage repair-deficient pancreatic cancer

47. Additional file 2: Figure S2. of PKM2 promotes tumor angiogenesis by regulating HIF-1Îą through NF-ÎşB activation

48. Additional file 3: Figure S3. of PKM2 promotes tumor angiogenesis by regulating HIF-1Îą through NF-ÎşB activation

49. Additional file 4: Figure S4. of PKM2 promotes tumor angiogenesis by regulating HIF-1α through NF-κB activation

50. Additional file 5: Figure S5. of PKM2 promotes tumor angiogenesis by regulating HIF-1α through NF-κB activation

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