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3. A novel Alzheimer disease locus located near the gene encoding tau protein.

4. Author Correction: Common variants in Alzheimer’s disease and risk stratification by polygenic risk scores (Nature Communications, (2021), 12, 1, (3417), 10.1038/s41467-021-22491-8)

6. Degenerazioni lobari frontotemporali: neuropatologia, genetica e classificazione

7. Cerebellar and subcortical atrophy contribute to psychiatric symptoms in frontotemporal dementia

8. Progress toward standardized diagnosis of vascular cognitive impairment: Guidelines from the Vascular Impairment of Cognition Classification Consensus Study

9. The Vascular Impairment of Cognition Classification Consensus Study

10. Motor symptoms in genetic frontotemporal dementia: developing a new module for clinical rating scales

11. New insights into the genetic etiology of Alzheimer's disease and related dementias

12. Protein network analysis reveals selectively vulnerable regions and biological processes in FTD

16. Genetic architecture of sporadic frontotemporal dementia and overlap with Alzheimerʼs and Parkinsonʼs diseases

20. A C6orf10/LOC101929163 locus is associated with age of onset in C9orf72 carriers

21. Common variants in Alzheimer’s disease and risk stratification by polygenic risk scores

22. Common variants in Alzheimer’s disease and risk stratification by polygenic risk scores

23. Common variants in Alzheimer's disease and risk stratification by polygenic risk scores

24. Common variants in Alzheimer's disease and risk stratification by polygenic risk scores

26. Démence sémantique : réflexions d’un groupe de travail pour des critères de diagnostic en français et la constitution d’une cohorte de patients

28. Alzheimer disease with cerebrovascular disease and vascular dementia: clinical features and course compared with Alzheimer disease

30. Correction to:A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity (Acta Neuropathologica, (2019), 138, 2, (237-250), 10.1007/s00401-019-02026-8)

31. Correction to: A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity (Acta Neuropathologica, (2019), 138, 2, (237-250), 10.1007/s00401-019-02026-8)

32. Mendelian randomization implies no direct causal association between leukocyte telomere length and amyotrophic lateral sclerosis

33. Progress toward standardized diagnosis of vascular cognitive impairment

38. Correction to: A nonsynonymous mutation in PLCG2 reduces the risk of Alzheimer’s disease, dementia with Lewy bodies and frontotemporal dementia, and increases the likelihood of longevity

39. A novel Alzheimer disease locus located near the gene encoding tau protein

40. Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates A beta, tau, immunity and lipid processing (vol 51, pg 414, 2019)

41. Nat Genet

42. Author Correction: Genetic meta-analysis of diagnosed Alzheimer’s disease identifies new risk loci and implicates Aβ, tau, immunity and lipid processing (Nature Genetics, (2019), 51, 3, (414-430), 10.1038/s41588-019-0358-2)

43. Genetic variation across RNA metabolism and cell death gene networks is implicated in the semantic variant of primary progressive aphasia

44. Author Correction: Genetic meta-analysis of diagnosed Alzheimer's disease identifies new risk loci and implicates Abeta, tau, immunity and lipid processing

46. Genetic variation across RNA metabolism and cell death gene networks is implicated in the semantic variant of primary progressive aphasia

47. Relations between C9orf72 expansion size in blood, age at onset, age at collection and transmission across generations in patients and presymptomatic carriers

48. CXCR4involvement in neurodegenerative diseases

49. Linguistic characteristics of genetic primary progressive aphasias: a retrospective study of 27 cases carrying GRN and c9orf72 mutations

50. Genetic risk for neurodegenerative disorders, and its overlap with cognitive ability and physical function

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