38 results on '"Fike, Adam J."'
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2. miR-21 promotes B cell autoreactivity, inflammatory signaling, and metabolism in TLR7-driven systemic autoimmunity
3. The native ORAI channel trio underlies the diversity of Ca2+ signaling events
4. Orai3 and Orai1 mediate CRAC channel function and metabolic reprogramming in B cells
5. Author response: Orai3 and Orai1 mediate CRAC channel function and metabolic reprogramming in B cells
6. The Post-GWAS Era: How to Validate the Contribution of Gene Variants in Lupus
7. The mitochondrial sodium/calcium exchanger NCLX (Slc8b1) in B lymphocytes
8. STAT3 signaling in B cells controls germinal center zone organization and recycling
9. Orai3 and Orai1 are essential for CRAC channel function and metabolic reprogramming in B cells
10. Context-Dependent miR-21 Regulation of TLR7-Mediated Autoimmune and Foreign Antigen–Driven Antibody-Forming Cell and Germinal Center Responses
11. STAT4 Is Largely Dispensable for Systemic Lupus Erythematosus–like Autoimmune- and Foreign Antigen–Driven Antibody-Forming Cell, Germinal Center, and Follicular Th Cell Responses
12. TLR7 Negatively Regulates B10 Cells Predominantly in an IFNγ Signaling Dependent Manner
13. Serine Phosphorylation of the STAT1 Transactivation Domain Promotes Autoreactive B Cell and Systemic Autoimmunity Development
14. Serine phosphorylation of the STAT1 transactivation domain promotes autoreactive B cell and systemic autoimmunity development
15. Type II but Not Type I IFN Signaling Is Indispensable for TLR7-Promoted Development of Autoreactive B Cells and Systemic Autoimmunity
16. Lung transcriptional unresponsiveness and loss of early influenza virus control in infected neonates is prevented by intranasal Lactobacillus rhamnosus GG
17. Strain-Dependent Contribution of MAVS to Spontaneous Germinal Center Responses
18. Dissecting the defects in the neonatal CD8+ T-cell response
19. Neonatal influenza-specific effector CTLs retain elevated CD31 levels at the site of infection and have decreased IFN-γ production
20. Dissecting the defects in the neonatal CD8+ T‐cell response.
21. The transcription factor T-bet is regulated by microRNA-155 in murine anti-viral CD8+ T cells via SHIP-1.
22. Long-Term Persistence of Exhausted CD8 T Cells in Chronic Infection Is Regulated by MicroRNA-155
23. Public Clonotypes and Convergent Recombination Characterize the Naïve CD8+ T-Cell Receptor Repertoire of Extremely Preterm Neonates
24. The Transcription Factor T-Bet Is Regulated by MicroRNA-155 in Murine Anti-Viral CD8+ T Cells via SHIP-1
25. Characterization of CD31 expression on murine and human neonatal T lymphocytes during development and activation
26. MicroRNA-155 regulates T-bet expression in anti-viral CD8+ T cells
27. Toll-Like Receptor (TLR)-2 Mediates Recruitment of Neutrophils During Influenza Infection in a Neonatal Murine Model
28. The CD8+ T-cell receptor (TCR) repertoire in extremely preterm neonates is highly shared and convergent
29. Dissecting the defects in the neonatal CD8+T‐cell response
30. Neonatal influenza‐specific effector CTLs retain elevated CD31 levels at the site of infection and have decreased IFN‐γ production.
31. Phosphatidylinositol 3-Kinase p110δ Isoform Regulates CD8+ T Cell Responses during Acute Viral and Intracellular Bacterial Infections
32. The Transcription Factor T-Bet is Regulated by MicroRNA-155 in Murine Anti-Viral CD8+ T Cells via SHIP-1.
33. Neonatal influenza‐specific effector CTLs retain elevated CD31 levels at the site of infection and have decreased IFN‐γ production
34. Characterization of CD31 expression on murine and human neonatal T lymphocytes during development and activation
35. Phosphatidylinositol 3-Kinase p110δ Isoform Regulates CD8+ T Cell Responses during Acute Viral and Intracellular Bacterial Infections.
36. Dissecting the defects in the neonatal CD8 + T-cell response.
37. Public Clonotypes and Convergent Recombination Characterize the Naïve CD8 + T-Cell Receptor Repertoire of Extremely Preterm Neonates.
38. The Transcription Factor T-Bet Is Regulated by MicroRNA-155 in Murine Anti-Viral CD8 + T Cells via SHIP-1.
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