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6. Abstract form for the Irish Journal of Medical Science v workshop on gastroduodenal pathology and Helicobacter pylori July 5th — 7th 1992 — Dublin, Ireland

Author

Logan, R. P. H., Gummett, P. A., Walker, M. M., Karim, Q. N., Baron, J. H., Misiewicz, J. J., Trieber, G., Walker, S., Klotz, U., Lozniewski, A., Weber, M., de Korwin, J. D., Floquet, J., Conroy, M. C., Burdin, J. C., Mannes, G. A., Bayerdörffer, E., Höchter, W., Weingart, J., Heldwein, W., Sommer, A., Müller-Lissner, S., Bomschein, W., Miehlke, S., Weinzierl, M., Ruckdeschel, G., von Wulffen, H., Köpcke, W., Stolte, M., Rune, S. J., Justesen, T., Hansen, J. M., Jensen, T. G., Eriksen, J., Thomsen, O. ø., Scheibel, J., Bonnevie, O., Bremmelgaard, A., Vilien, M., Knuhtsen, S., Elsborg, L., Hansen, J., Lauritsen, K., Wulff, H. R., Boixeda, D., Ballestero, S., Cantón, R., De Rafael, L., Martinm de Argila, C., Pozuelo, M. J., Sampedro, J., Baquero, F., Grigoriev, P. Ya., Isakov, V. A., Iakovenko, E. P., Hirschl, A. M., Brandstätter, G., Dragosics, B., Hentschel, E., Kundi, M., Rotter, M. L., Schütze, K., Taufer, M., Neri M, Susi D, Bovani I, Pindo R, Cuccurullo F., Coelho, L. G. V., Passos, M. C. F., Chausson, Y., Vieira, W. L. S., Castro, F. J., Franco, J. M. M., Fernandes, M. L. M., Castro, L. P., Jonas, C., De Koster, E., Van Gossum, M., Depierreux, M., Cheval, M., Deltenre, M., Schütz, E., Bethke, B., Lee, A., Hegedus, E., O’Rourke, J., Larsson, H., Sjöstedt, S., Veress, B., Nord, C. E., Sobala, G. M., George, R., Tompkins, D., Finlay, J., Manning, A., Sant, S., Xia, H. X., Daw, M., Gilvarry, J., Keane, C. T., O’Morain, C., Rubio, M. A., Hegarty, B., Blum, A. L., Sulser, E., Stadelmann, O., Munoz, N., Buiatti, E., Vivas, J., Oliver, W., Cano, E., Peraza, S., Castro, D., Sanchez, V., Andrade, O., Benz, M., Mendz, G. L., Hazell, S. L., Salmela, K. S., Roire, R. P., Hook-Nikanne, J., Kosunen, T. U., Salaspur, M., Luke, C. J., Reynolds, D. D. J., Penn, C. W., Bode, G., Mauch, F., Ditschuneit, H., Malfertheiner, P., Ferrero, Richard L., Agnes Labigne, Eaton, K. A., Krakowka, S., Mobley, H. L. T., Hu, Li-Tai, Foxall, P. A., Moran, A. P., Helander, I. M., Altman, C., Sobhani, I., Vissugaire, C., Migrant, M., Etienne, J. P., Sommi, P., Ricci, V., Fiocca, R., Cova, E., Figura, N., Romano, M., Ivey, K. J., Solcia, E., Ventura, U., Nilius, M., Schieffer, S., Hengels, K. J., Jablonowski, H., Strohmeyer, G., Cabrai, M. D., Barbosa, A. J. A., Lima Hr., G. F., Oliveira, C. A., Polak, J. M., Oderda, G., Villani, L., Altare, F., Morra, I., Miserendino, L., Ansaldi, N., Dixon, M. F., Wyatt, J. I., Axon, A. T. R., Beattie, S., Hamilton, H., Shabib, S., Cutz, E., Drumm, B., Sherman, P., Noach, L. A., Rolf, T., Bosma, N. B., Schwartz, M. P., Oosting, J., Rauws, E. A. J., Tytgat, G. N. J., Andrew, A., Nardone, G., d’Ormiento, F., Pontillo, M., Lobo, A. J., Uff, J. S., McNulty, C. N. M., Wilkinson, S. P., Suriani, R., Pallante, C., Ravizza, M., Galliano, D., Sallio, D., Malandrino, M., Oneglio, R., Colozza, M., Mazzucco, D., Gaia, E., Eidt, S., Vincent, P., Gottrand, F., Turck, D., Lecomte-Houcke, M., Leclerc, H., Bonvicini, F., Pretolani, S., Baraldini, M., Cilla, D., Baldinelli, S., Bazocchi, E., Acampora, P., Careddu, N., Brocchi, E., Gasbarrini, G., Joubert, M., Bazin, N., Thiaucourt, D., Protte, E., Gissler, C., Duprez, A., Merlin, P., Forestier, S., Labenz, J., Gyenes, E., Rühl, G. H., Börsch, G., Daskalopoulos, G., Carrick, J., Lian, R., Wagner, S., Bleck, J., Gebel, M., Bär, W., Manns, M., Lamouliatte, H., Bernard, P. H., Cayla, R., Vialette, G., Quinton, A., Mégraud, F., Lemaire, M., Quinten, A., De Mascarel, A., Webb, P., Forman, D., Knight, T., Wilson, A., Graves, S., Newell, D., Elder, J., Tonelli, E., Gatte, M. R. A., Ghironzi, G. C., Giulianelli, G., Bamford, K. B., Collins, J. S. A., Bickley, J., Johnston, B. T., Potts, S., Boston, V., Owen, R. J., Sloan, J., Basso, L., Lawlor, S., Clune, J., Szelényi, H., Stohmeyer, G., Macedo, G., Iglésias, I., Chaves, A. P., Loureiro, A., Katelaris, P. H., Seow, F., Lin, B., Napoli, J., Hones, D. B., Ngu, M. C., Akopyantz, Natalia S., Bukanov, Nikolay O., Westblom, T. Ulf, Berg, Douglas E., Nyst, J. F., Denis, P., Buset, M., De Reuck, M., Nielsen, H., Andersen, L. P., Birkholz, Sabine, Knipp, Ulrich, Nietzki, Claudia, Opferkuch, Wolfgang, Crabtree, J. E., Peichl, P., Lindly, I. J. D., Deusch, K., Seifirth, C., Funk, A., Dahie, I., Reut, K., Classen, M., Gionchetti, P., Vaira, D., Campieri, M., Bertinelli, E., Menegatti, M., Belluzzi, A., Briognola, C., Miglioli, M., Barbara, L., Tommaso, A. Di, Magistris, M. T. De, Bugnoli, M., Petracca, R., Covacci, A., Censini, S., Rappuoli, R., Abrignani, S., Territo, M. C., Smela, K. L., Reeve, J. R., Lee, T. D., Walsh, J. H., Armellini, D., Crabtree, J. E., Xiang, Z. Y., Mitchell, H. M., Hu, P. J., Li, Y. Y., Wang, Z. J., Zhao, S. M., Liu, Q., Chen, M., Du, G. G., Filipe, M. I., Reed, P. I., Craanen, M. E., Blok, P., Dekker, W., Colombo, E., Redaelli, D., Santangelo, M., Spinelli, M., Farinati, F., Valiante, F., Delia Libera, G., Germanà, B., Baffa, R., Rugge, M., Vianelo, F., Di Mario, F., Sipponen, Pentti, Rokkas, T., Popotheodorou, G., Kaldgeropoulos, N., Deprez, C., Galand, P., Fox, J. G., Wishnok, P., Murphy, J. C., Tannenbaum, S., Correa, P., Parsonnet, Julie, Macor, C., Da Broi, G. L., Avellinio, C., Reifen, R., Rasooly, I., Millson, M. E., Murphy, K., Thomas, J. E., Eastham, E. J., Malorgio, E., Dell’Olio, D., Kemmer, T. P., Dominguez-Munoz, J. E., Klingel, H., Gatto, M. R. A., Olivieri, R., Bayeli, R. F., Abate, L., De Gregorio, L., Aziz, J., Esposito, E., Basagni, C., Guilluy, R., Rousseau-Tsangaris, M., Brazier, J. L., Wadstiöm, Torkel, Tyszkiewicz, Tadeusz, Bergenzaun, Per, Olsson, Karin, Birac, C., Tall, F., Albenque, M., Labigne, A., Megraud, F., Feldman, R. A., Deeks, J., Glupczynski, Y., Burette, A., Goossens, H., Van den Boore, C., Butzler, J. P., Veldhuyzen van Zanten, S., Best, L., Benzanson, G., Haldane, D., Hazell, S., Mapstone, N. P., Lynch, D. A. F., Quirke, P., Taylor, D. E., Chang, N., Eaton, M., Stockdale, E., Salama, S. M., Thompson, L., Cockayne, A., Spiller, R. C., Leen, E., Sweeney, E., Klann, H., Hatz, R., Bornschein, W., Simon, T., Eimiller, A., Bolle, F., Schweikert, C., Köpeke, W., Moss, S. F., Bishop, A. E., Calam, J., Cahill, R. J., Xia, H., Solnick, J., and Tompkins, L.

Published
1992
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17. [Phosphorus NMR spectroscopy. Its value in the diagnosis of metabolic myopathies. A case of Mac Ardle's disease]

Author

Kaminsky P, Mariarosa AB Melone, Brunotte F, Jm, Escanye, Robin B, Floquet J, Ml, Duc, Robert J, Duc M, Kaminsky, P, Melone, Mariarosa Anna Beatrice, Brunotte, F, Escanye, Jm, Robin, B, Floquet, J, Duc, Ml, Robert, J, and Duc, M.

Subjects
Adult, Magnetic Resonance Spectroscopy, Muscular Diseases, Reference Values, Glycogen Storage Disease Type V, Humans, Female, Phosphorus, Isoelectric Point
Abstract

Phosphorus nuclear magnetic resonance spectroscopy is a non-invasive method used to study muscle bioenergetics in vivo. A new case of Mc Ardle's disease (myophosphorylase deficiency) is reported here. In a context of metabolic myopathy this method can provide a diagnosis of glycogenosis. The spectra obtained at exercise and during recovery determine the degree of enzyme deficiency with satisfactory precision.

Published
1990

18. Influence of muramyl dipeptide on established experimental arthritis in rats

Author

Jouzeau, J, Drelon, E, Gillet, G., Bannwarth, B, Fener, P., Charriere, G., Payan, E., Chauvelot-Moachon, L, Batt, A, Floquet, J., Laboratoire de Physiopathologie et Pharmacologie Articulaire (CNRS URA 1288), Service de Rhumatologie [CHRU Nancy], Centre Hospitalier Régional Universitaire de Nancy (CHRU Nancy), Institut de biologie et chimie des protéines [Lyon] (IBCP), Université Claude Bernard Lyon 1 (UCBL), Université de Lyon-Université de Lyon-Centre National de la Recherche Scientifique (CNRS), Laboratoire de Pharmacologie (URA CNRS 595 ), Centre du Médicament [Nancy], Université Henri Poincaré - Nancy 1 (UHP), and Service d’Anatomie Pathologique [CHRU Nancy]

Subjects
Dose-Response Relationship, Drug, [SDV]Life Sciences [q-bio], Rats, Inbred WF, Arthritis, Experimental, Rats, Radiography, body regions, [SDV.MHEP.RSOA]Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal system, Immunoglobulin G, parasitic diseases, Animals, Female, Acetylmuramyl-Alanyl-Isoglutamine, [SDV.MHEP]Life Sciences [q-bio]/Human health and pathology, Glycoproteins
Abstract

International audience; The effects of MDP, a potent inducer of cytokines, were studied in four batches of Wistar Furth rats with established experimental arthritis. Arthritic rats were given a daily sc injection of 10, 100, 200 or 400 micrograms MDP respectively. Muramyl dipeptide increased the severity of clinical events in a dose-dependent manner, with the exception of the 10 micrograms dose which was ineffective. The levels of anti-collagen antibodies were not however significantly enhanced by MDP. Radiological lesions and histological changes were maximal at high dosage regimens. Paradoxically, the acute phase reactive alpha 1 glycoprotein was little affected by MDP treatment.

Published
1991

34. Atrophies systématisées multiples, arriération mentale, amyotrophie neurogène et fragilité osseuse congénitale

Author

A. Bajolle, Pierson M, Neimann N, Floquet J, Martin Jj, and Vidailhet M

Subjects
Subluxation, medicine.medical_specialty, Pediatrics, business.industry, Osteoporosis, Neurological disorder, medicine.disease, Spinal cord, Amyotrophy, Surgery, White matter, Peripheral neuropathy, medicine.anatomical_structure, Neurology, medicine, Neurology (clinical), business, Pathological
Abstract

Five cases of a congenital neurological disorder are reported. Four patients, born after a breech delivery, belong to one sibship while the fifth patient is the only child in another family. The clinical features include quadriplegia, amyotrophy, a peripheral neuropathy, severe mental retardation and a subluxation of the hips. X-rays reveal diffuse osteoporosis and multiple spontaneous fractures. Autopsies in 3 patients showed multiple system atrophies involving the spinal cord and the cerebellum, coarse cerebral gyri and a marked reduction in volume of the white matter. These various pathological features are compared with the lesions found in a few other cases reported in the literature, none of which can be considered to be identical to the ones described. It is therefore felt that the condition under discussion represents a new syndrome to be classified, at least temporarily, within the group of multiple system atrophies.

Published
1976
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35. Microvascularization of the intracranial dura mater

Author

A Czorny, Serge Bracard, Floquet J, P Forlodou, Bernard C, J M Race, J. Roland, and L. Picard

Subjects
musculoskeletal diseases, Adult, Dura mater, Venous lumen, Pathology and Forensic Medicine, Veins, Cranial vault, Medicine, Humans, Radiology, Nuclear Medicine and imaging, Fibrous layer, Plexus, integumentary system, business.industry, Microcirculation, Arterial plexus, Meninges, Anatomy, musculoskeletal system, Meningeal Arteries, nervous system diseases, Radiography, medicine.anatomical_structure, nervous system, Surgery, Dura Mater, business
Abstract

The authors have studied the microvascularization of the intracranial dura mater by microradiographic and histologic examination of 73 injected anatomic specimens. There exists a very abundant superficial arterial plexus which also serves to supply the inner table of the cranial vault. This plexus is continuous, even at the walls of the venous sinuses and the dural septa. The arteries are for the most part tortuous. The veins may be satellites of the arteries or, on the contrary, from a plexiform network passing through crevices in the interior of the dural layer. The walls of these veins consist only of an endothelium to be seen within the fibrous layer of the dura. Often, the arteries compress the venous lumen; this dangerous situation probably explains the frequent occurrence of arteriovenous fistulae of the dura mater, known by the name of dural fistulae.

Published
1987

36. HIGH-FIELD MAGNETIC-RESONANCE-IMAGING (MRI) OF THE RABBIT KNEE

Author

Paola Peiffert, Pierre GILLET, Paul Walker, Gegout, P., Jean Marie Escanyé, Floquet, J., Jacques Robert, Patrick Netter, Alain Gaucher, Service de Médecine Nucléaire [Nancy], Centre Hospitalier Régional Universitaire de Nancy ( CHRU Nancy ), Physiopathologie, Pharmacologie et Ingénierie articulaires ( PPIA ), Université Henri Poincaré - Nancy 1 ( UHP ) -Centre National de la Recherche Scientifique ( CNRS ), Laboratoire Electronique, Informatique et Image ( Le2i ), Université de Bourgogne ( UB ) -Centre National de la Recherche Scientifique ( CNRS ) -AgroSup Dijon - Institut National Supérieur des Sciences Agronomiques, de l'Alimentation et de l'Environnement, laboratoire de methodologie RMN, Centre Hospitalier Régional Universitaire de Nancy (CHRU Nancy), Physiopathologie, Pharmacologie et Ingénierie articulaires (PPIA), Université Henri Poincaré - Nancy 1 (UHP)-Centre National de la Recherche Scientifique (CNRS), Laboratoire Electronique, Informatique et Image [UMR6306] (Le2i), Université de Bourgogne (UB)-Centre National de la Recherche Scientifique (CNRS)-École Nationale Supérieure d'Arts et Métiers (ENSAM), Arts et Métiers Sciences et Technologies, HESAM Université (HESAM)-HESAM Université (HESAM)-Arts et Métiers Sciences et Technologies, HESAM Université (HESAM)-HESAM Université (HESAM)-AgroSup Dijon - Institut National Supérieur des Sciences Agronomiques, de l'Alimentation et de l'Environnement, Walker, Paul, Université de Bourgogne (UB)-École Nationale Supérieure d'Arts et Métiers (ENSAM), and HESAM Université (HESAM)-HESAM Université (HESAM)-AgroSup Dijon - Institut National Supérieur des Sciences Agronomiques, de l'Alimentation et de l'Environnement-Centre National de la Recherche Scientifique (CNRS)

Subjects
[ INFO.INFO-IM ] Computer Science [cs]/Medical Imaging, [INFO.INFO-IM] Computer Science [cs]/Medical Imaging, [INFO.INFO-IM]Computer Science [cs]/Medical Imaging, ComputingMilieux_MISCELLANEOUS
Abstract

International audience

37. Auto-immune spondylodiscitis associated with collagen induced arthritis in rats: high field MRI findings

Author

Pierre GILLET, Fener, P., Escanyé, J., Walker, P., Bannwarth, B., Jouzeau, J., Drelon, E., Floquet, J., Netter, P., Robert, J., Laboratoire de Physiopathologie et Pharmacologie Articulaire (CNRS URA 1288), Service de Rhumatologie [CHRU Nancy], Centre Hospitalier Régional Universitaire de Nancy (CHRU Nancy), Service de Médecine Nucléaire [Nancy], and Service d’Anatomie Pathologique [CHRU Nancy]

Subjects
Tail, Discitis, [SDV]Life Sciences [q-bio], Rats, Inbred WF, Arthritis, Experimental, Magnetic Resonance Imaging, Autoimmune Diseases, Rats, Radiography, [SDV.MHEP.RSOA]Life Sciences [q-bio]/Human health and pathology/Rhumatology and musculoskeletal system, Animals, Female, Collagen, [SDV.MHEP]Life Sciences [q-bio]/Human health and pathology
Abstract

International audience; The spinal involvement of the tail was studied in Wistar Furth rats immunized with bovine native type II collagen. Focal caudal autoimmune spondylodiscitis occurred 5 weeks after sensitization, as assessed histopathologically. High field Magnetic Resonance Imaging (MRI) was useful in depicting these caudal abnormalities that were related to juxta-diskal enthesitis. The occurrence of such inflammatory enthesopathies could serve as an experimental approach for physiopathological and therapeutical studies of spondylarthropathies.

42. Adynamie épisodique héréditaire

Author

Van Bogaert, L., primary, Kissel, P., additional, Schmitt, J., additional, Martin, J.J., additional, Claes, C., additional, Duc, M., additional, Royer, R., additional, Barrucand, D., additional, Grignon, G., additional, and Floquet, J., additional

Published
1967
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43. Cyst formation in gastric heterotopic pancreas: report of two cases

Author

Floquet J, M A Bigard, A L Verain, P Poisson, Boissel P, Michel Claudon, and Régent D

Subjects
Adult, Male, medicine.medical_specialty, Pathology, Radiography, Stomach Diseases, Computed tomography, Choristoma, Stomach Neoplasms, medicine, Humans, Radiology, Nuclear Medicine and imaging, Pancreas, Gastric wall, Ultrasonography, medicine.diagnostic_test, Cysts, business.industry, digestive, oral, and skin physiology, Ultrasound, digestive system diseases, Cyst formation, Radiology, Tomography, X-Ray Computed, business, Heterotopic pancreas
Abstract

Cyst formation is an uncommon pathologic finding in heterotopic pancreas. It is sometimes reported in a duodenal location but very rarely in a gastric location and leads to very large lesions of unusual endoscopic and radiographic appearance. Two cases of gastric heterotopic pancreas are described in which cystic zones in the gastric wall were found at preoperative ultrasound or computed tomography.

Published
1988
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46. [112 cases of sporadic and genetically determined pheochromocytoma: a comparative pathologic study].

Author

Gosset P, Lecomte-Houcke M, Duhamel A, Labat-Moleur F, Patey M, Floquet J, Viennet G, Berger-Dutrieux N, Caillou B, and Franc B

Subjects
Adrenal Cortex pathology, Adult, Diagnosis, Differential, Female, Humans, Hyperplasia, Male, Multiple Endocrine Neoplasia Type 2a genetics, Multiple Endocrine Neoplasia Type 2a pathology, Multiple Endocrine Neoplasia Type 2b genetics, Multiple Endocrine Neoplasia Type 2b pathology, Neurofibromatosis 1 genetics, Neurofibromatosis 1 pathology, von Hippel-Lindau Disease genetics, von Hippel-Lindau Disease pathology, Adrenal Gland Neoplasms genetics, Adrenal Gland Neoplasms pathology, Pheochromocytoma genetics, Pheochromocytoma pathology
Abstract

The aim of this study was to compare 64 genetically determined pheochromocytomas (PH) (49 MEN IIa, 3 MEN IIb, 6 Von Recklinghausen diseases, 1 von Hippel-Lindau disease, 5 familial pheochromocytomas) and 48 sporadic PH. Genetically determined PH were more often observed among men and more frequently bilateral and multicentric than sporadic PH. Sporadic tumors had more often adrenal capsular invasion, necrosis and pseudocysts. Genetically determined PH were more differentiated with an insular pattern, hyaline globules and a higher percentage of polyhedric cells. Sporadic tumors were less differentiated with more frequently a diffuse pattern and small cells. Adrenal medullar hyperplasia was significantly associated with genetically determined PH. Adrenal cortical hyperplasia was not associated with a particular type of PH. The PS100 and chromogranin immunodetection was equivalent in both groups.

Published
1999

47. Gerstmann-Sträussler-Scheinker disease and the French-Alsatian A117V variant.

Author

Mohr M, Tranchant C, Steinmetz G, Floquet J, Grignon Y, and Warter JM

Subjects
Adult, Amino Acid Substitution, Female, France, Gerstmann-Straussler-Scheinker Disease pathology, Humans, Male, Pedigree, Genetic Variation, Gerstmann-Straussler-Scheinker Disease genetics, Point Mutation, Polymorphism, Genetic
Abstract

Gerstmann-Sträussler-Scheinker disease is a rare familial form of prion disease. This autosomal dominant disorder is constantly associated with a point mutation on the PrP gene. Eight mutations affecting respectively codons 102, 105, 117, 145, 202, 212 and 218, have been so far described. Symptoms are variable and include ataxia and dementia. They generally appear between the fourth and sixth decade. Mean duration of the disease (5 years) is on the whole longer than that of other familial forms of prion diseases. Gerstmann-Sträussler-Scheinker disease is neuropathologically characterized by the presence of numerous multicentric or unicentric PrP amyloid deposits widespread throughout the encephalon. Spongiform change is inconstant. Neurofibrillary tangles have been described in some families. Clinicopathological features show considerable variability. Pathogenesis of amyloidosis and associated lesions as well as factors underlying the phenotypic polymorphism of the disease remain only partially known.

Published
1999

48. Influence of a high-calcium carbonate diet on the incidence of experimental colon cancer in rats.

Author

Quilliot D, Belbraouet S, Pelletier X, Guéant JL, Floquet J, and Debry G

Subjects
Animals, Colonic Neoplasms epidemiology, Feces chemistry, Female, Hydrogen-Ion Concentration, Incidence, Laminin blood, Rats, Rats, Wistar, Calcium Carbonate pharmacology, Colonic Neoplasms diet therapy, Diet
Abstract

This study was done to determine whether a high dietary calcium carbonate concentration could protect against colon tumors in rats. Female Wistar rats were randomly assigned to one of four groups and maintained on an 8% lipid diet for an adaptation period of four weeks. All groups were then fed a 24% lipid diet (sunflower oil), with (Groups 2 and 4) or without (Groups 1 and 3) a 1.5% calcium carbonate supplement. They were intrarectally instilled with saline (Groups 1 and 2) or nitrosomethylurea (NMU) (Groups 3 and 4). Fecal sterol output and pH were analyzed for one week each month. Histological analysis was done at the end of the 32-week experiment. No tumors were found in the non-NMU-treated animals. The NMU-treated rats had tumors: 31% in Group 3 and 30% in Group 4. The calcium carbonate supplement had no effect on this incidence. The lipid and cholesterol excretions of the calcium carbonate-supplemented rats were significantly enhanced. The coprostanol output was not altered, although its fecal concentration of the calcium-supplemented rats was decreased. Although neither lipid overload nor NMU treatment altered the fecal pH, it was significantly increased in both calcium carbonate-supplemented groups. These findings suggest that additional calcium as carbonate has no effect on colon tumor incidence, although the fecal composition is altered. The increased pH of the feces due to the carbonate could have the opposite effect to calcium.

Published
1999
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49. Medullary thyroid carcinoma: search for histological predictors of survival (109 proband cases analysis).

Author

Franc B, Rosenberg-Bourgin M, Caillou B, Dutrieux-Berger N, Floquet J, Houcke-Lecomte M, Justrabo E, Lange F, Labat-Moleur F, Le Bodic MF, Patey M, Beauchet A, Saint-André JP, Hejblum G, and Viennet G

Subjects
Adolescent, Adult, Aged, Aged, 80 and over, Child, Female, Humans, Male, Middle Aged, Retrospective Studies, Survival Analysis, Carcinoma, Medullary mortality, Carcinoma, Medullary pathology, Thyroid Neoplasms mortality, Thyroid Neoplasms pathology
Abstract

A group of 13 pathologists belonging to the French Calcitonin Tumor Study Group (GETC: Groupe d'Etude des Tumeurs à Calcitonine) examined the histological slides and medical records of 109 proband cases of medullary thyroid carcinoma (MTC) diagnosed on clinical features. The cases belonged to the various forms of the disease (80 sporadic and 29 familial MTC). The aim of the study was to detect histological predictors for survival by comparing morphological data from patients killed by the disease versus the others. Twenty-seven histological parameters were considered, including cellular heterogeneity, shape of the cells, and cytoplasmic characteristics. Other parameters such as sex, age, and phenotype of the disease were also studied. First, predictive parameters of interest on survival function were selected by univariate analysis (Mantel-Cox test). Then, the extracted parameters were tested in a multifactorial analysis using the Cox's forward stepping proportional hazard model. Five parameters were significantly associated with a lower survival function: presence of necrosis in the tumor (P = .001), squamous pattern (P = .002), age over 45 years (P = .004), presence of oxyphil cells in the tumor and absence of cells with intermediate cytoplasm (P = .025), less than 50% of calcitonin immunoreactive cells in the tumor (P = .04).

Published
1998
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50. [Extra-cardiac rhabdomyoma of the fetal type].

Author

Grignon Y, Baylac F, Nodari F, Maury F, Chastagner P, Plénat F, and Floquet J

Subjects
Humans, Infant, Male, Fetal Diseases pathology, Muscle Neoplasms pathology, Rhabdomyoma pathology
Abstract

Rhabdomyomas are benign tumors of striated muscle. They are distinguished by topographic data: cardiac or genital and by histological criteria: foetal or adult type. The foetal type is the most heterogeneous, with either a majority of immature cells or a mixture of maturing elements. This diversity has led to distinguish immature (or standard) foetal Rhabdomyomas from intermediate foetal Rhabdomyomas. One observation of this last type is reported. The authors highlight the essential characteristics of foetal Rhabdomyomas, however the exact meaning remains unclear: an anomaly in the differentiation of the striated muscle? or a true tumoral process through genetic anomaly?

Published
1997

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