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3 results on '"MKEY"'

1. Design and implementation of the company mobile office system based on the Mkey middleware.

Author

Chen, Qingzhang, Li, Xinghua, Qian, Cheng, and Yang, Chen

Abstract

With the development of mobile technology and the expansion of business, traditional management information system in the hydropower enterprise which relies on fixed office space and equipment has been unable to meet current work needs. This paper presents an enterprise mobile office system based on Mkey middleware, which has break the space-temporal manacle and can solve many operating systems' compatibility issues of mobile phone. It realized the integration of Internet, communication technology and embedded operating system. This system modeled the operating habits and functional characteristics after the computer. It is easy to use. The user can use it skillfully and flexibly without any additional training. It solves the problem that the inconvenience of carrying a laptop. And it improves the response speed and the stability of the system. With this system, the company managers, designers can approve documents, browse notice, handle personal affairs at anywhere and anytime, and they can freely associated with the main management information system. [ABSTRACT FROM PUBLISHER]

Published
2012
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2. Molecular Biology of Atherosclerotic Ischemic Strokes

Author

Maria Chiara Velardo, Antonino Tuttolomondo, Maria Grazia Puleo, Antonio Pinto, Francesca Corpora, Mario Daidone, Tuttolomondo A., Puleo M.G., Velardo M.C., Corpora F., Daidone M., and Pinto A.

Subjects
0301 basic medicine, Inflammasomes, Cerebral arteries, microglia, Disease, Review, neuroinflammation, Brain ischemia, lcsh:Chemistry, 0302 clinical medicine, atherosclerosi, Stroke, lcsh:QH301-705.5, Spectroscopy, microRNA, General Medicine, MKEY, DKK-3, Computer Science Applications, microRNAs, Blood-Brain Barrier, Cardiology, medicine.symptom, Dectin-1, medicine.medical_specialty, Ischemia, Brain damage, Catalysis, Inorganic Chemistry, 03 medical and health sciences, Internal medicine, Diabetes mellitus, medicine, ischemic stroke, Animals, Humans, Physical and Theoretical Chemistry, Molecular Biology, business.industry, Organic Chemistry, AF, medicine.disease, Immunity, Innate, NLRP3 inflammasome, 030104 developmental biology, lcsh:Biology (General), lcsh:QD1-999, atherosclerosis, business, BBB, 030217 neurology & neurosurgery, Dyslipidemia, CD200-CD200R
Abstract

Among the causes of global death and disability, ischemic stroke (also known as cerebral ischemia) plays a pivotal role, by determining the highest number of worldwide mortality, behind cardiomyopathies, affecting 30 million people. The etiopathogenetic burden of a cerebrovascular accident could be brain ischemia (~80%) or intracranial hemorrhage (~20%). The most common site when ischemia occurs is the one is perfused by middle cerebral arteries. Worse prognosis and disablement consequent to brain damage occur in elderly patients or affected by neurological impairment, hypertension, dyslipidemia, and diabetes. Since, in the coming years, estimates predict an exponential increase of people who have diabetes, the disease mentioned above constitutes together with stroke a severe social and economic burden. In diabetic patients after an ischemic stroke, an exorbitant activation of inflammatory molecular pathways and ongoing inflammation is responsible for more severe brain injury and impairment, promoting the advancement of ischemic stroke and diabetes. Considering that the ominous prognosis of ischemic brain damage could by partially clarified by way of already known risk factors the auspice would be modifying poor outcome in the post-stroke phase detecting novel biomolecules associated with poor prognosis and targeting them for revolutionary therapeutic strategies.

Published
2020

3. Molecular Biology of Atherosclerotic Ischemic Strokes.

Author

Tuttolomondo, Antonino, Puleo, Maria Grazia, Velardo, Maria Chiara, Corpora, Francesca, Daidone, Mario, and Pinto, Antonio

Subjects
*CEREBRAL ischemia, *STROKE, *DISABILITIES, *OLDER patients, *CEREBRAL arteries, *ISCHEMIC preconditioning, *MOLECULAR biology, *IMPLANTABLE cardioverter-defibrillators
Abstract

Among the causes of global death and disability, ischemic stroke (also known as cerebral ischemia) plays a pivotal role, by determining the highest number of worldwide mortality, behind cardiomyopathies, affecting 30 million people. The etiopathogenetic burden of a cerebrovascular accident could be brain ischemia (~80%) or intracranial hemorrhage (~20%). The most common site when ischemia occurs is the one is perfused by middle cerebral arteries. Worse prognosis and disablement consequent to brain damage occur in elderly patients or affected by neurological impairment, hypertension, dyslipidemia, and diabetes. Since, in the coming years, estimates predict an exponential increase of people who have diabetes, the disease mentioned above constitutes together with stroke a severe social and economic burden. In diabetic patients after an ischemic stroke, an exorbitant activation of inflammatory molecular pathways and ongoing inflammation is responsible for more severe brain injury and impairment, promoting the advancement of ischemic stroke and diabetes. Considering that the ominous prognosis of ischemic brain damage could by partially clarified by way of already known risk factors the auspice would be modifying poor outcome in the post-stroke phase detecting novel biomolecules associated with poor prognosis and targeting them for revolutionary therapeutic strategies. [ABSTRACT FROM AUTHOR]

Published
2020
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