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2. Modeling gene × environment interactions in PTSD using human neurons reveals diagnosis-specific glucocorticoid-induced gene expression

5. Integrating deep learning and unbiased automated high-content screening to identify complex disease signatures in human fibroblasts

6. STRESS IN A DISH: MODELING THE IMPACT OF COMMON GENETIC VARIATION ON STRESS RESPONSE IN HIPSC-DERIVED NEURONS IN PTSD

8. Deficiency in prohormone convertase PC1 impairs prohormone processing in Prader-Willi syndrome

9. T73. MODELING GENE BY ENVIRONMENT INTERACTIONS IN POST-TRAUMATIC STRESS DISORDER USING HIPSC-DERIVED NEURONS

11. A human iPSC-array-based GWAS identifies a virus susceptibility locus in the NDUFA4 gene and functional variants

17. Reactive Astrocytes Derived From Human Induced Pluripotent Stem Cells Suppress Oligodendrocyte Precursor Cell Differentiation

20. A dual SHOX2:GFP; MYH6:mCherry knockin hESC reporter line for derivation of human SAN-like cells

21. Stem cell-derived neurons reflect features of protein networks, neuropathology, and cognitive outcome of their aged human donors

22. MODELING GENE X ENVIRONMENT INTERACTIONS IN PTSD USING GLUCOCORTICOID-INDUCED TRANSCRIPTOMICS IN HUMAN NEURONS

24. Nuclear genome transfer in human oocytes eliminates mitochondrial DNA variants

26. List of Contributors

28. Human oocytes reprogram somatic cells to a pluripotent state

35. Lipid Deprivation Induces a Stable, Naive-to-Primed Intermediate State of Pluripotency in Human PSCs

37. Additional file 1: Table S1. of CRISPR/Cas9-Correctable mutation-related molecular and physiological phenotypes in iPSC-derived Alzheimerâ s PSEN2 N141I neurons

38. Mitral valve reconstruction

44. Catalytic, asymmetric [alpha]-fluorination of acid chlorides: dual metal-ketene enolate activation

45. A surprising mechanistic 'switch' in Lewis acid activation: a bifunctional, asymmetric approach to [alpha]-hydroxy acid derivatives

46. Bifunctional asymmetric catalysis: cooperative Lewis acid/base systems

47. An asymmetric, bifunctional catalytic approach to non-natural [alpha]-amino acid derivatives

48. CRISPR/Cas9-Correctable mutation-related molecular and physiological phenotypes in iPSC-derived Alzheimer’s PSEN2 N141I neurons

50. Deficiency in prohormone convertase PC1 impairs prohormone processing in Prader-Willi syndrome

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