232 results on '"Ritter, Robert C."'
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2. CCK-sensitive C fibers activate NTS leptin receptor-expressing neurons via NMDA receptors.
3. Chemogenetic activation of ventral medullary astrocytes enhances feeding and corticosterone release in response to mild glucoprivation
4. Chemogenetic activation of ventral medullary astrocytes enhances feeding and corticosterone release in response to mild glucoprivation.
5. Vagal Afferent Signaling and the Integration of Direct and Indirect Controls of Food Intake
6. Gastrointestinal Peptides and the Control of Food Intake ☆
7. Linking drug and food addiction via compulsive appetite
8. Frequency-dependent facilitation of synaptic throughput via postsynaptic NMDA receptors in the nucleus of the solitary tract
9. Control of Meal Size by Central Noradrenergic Action
10. Blockade of hindbrain NMDA receptors containing NR2 subunits increases sucrose intake
11. NMDA receptor blockade attenuates CCK-induced reduction of real feeding but not sham feeding
12. Vagal Afferent NMDA Receptors Modulate CCK-Induced Reduction of Food Intake Through Synapsin I Phosphorylation in Adult Male Rats
13. Cholecystokinin activates both A- and C-type vagal afferent neurons
14. Cholinergic neurotransmission participates in increased food intake induced by NMDA receptor blockade
15. Daily CCK injection enhances reduction of body weight by chronic intracerebroventricular leptin infusion
16. CCK-Induced Reduction of Food Intake and Hindbrain MAPK Signaling Are Mediated by NMDA Receptor Activation
17. Neuroanatomy and Physiology of Abdominal Vagal Afferents
18. Leptin Sensitizes NTS Neurons to Vagal Input by Increasing Postsynaptic NMDA Receptor Currents
19. Increased Hypothalamic Signal Transducer and Activator of Transcription 3 Phosphorylation after Hindbrain Leptin Injection
20. Reduced sensitivity to the satiation effect of intestinal oleate in rats adapted to high-fat diet
21. Long-term CCK-leptin synergy suggests a role for CCK in the regulation of body weight
22. N-methyl-D-aspartate receptor subunit phenotypes of vagal afferent neurons in nodose ganglia of the rat
23. Nuclear Fos immunoreactivity in guinea pig myenteric neurons following activation of motor activity
24. An invitation for lawyers' participation in civil justice reform.
25. Intestinal infusions of oleate and glucose activate distinct enteric neurons in the rat
26. Glucoreceptors Controlling Feeding and Blood Glucose: Location in the Hindbrain
27. Cholecystokinin activates specific enteric neurons in the rat small intestine
28. Intestinal capsaicin transiently attenuates suppression of sham feeding by oleate
29. Caudal hindbrain participation in the suppression of feeding by central and peripheral bombesin
30. Suppression of sham feeding by intraintestinal nutrients is not correlated with plasma cholecystokinin elevation
31. NPY2 receptor activation in the dorsal vagal complex increases food intake and attenuates CCK-induced satiation in male rats
32. Reduced hindbrain and enteric neuronal response to intestinal oleate in rats maintained on high-fat diet
33. Diminished satiation in rats exposed to elevated levels of endogenous or exogenous cholecystokinin
34. NMDA receptor participation in control of food intake by the stomach
35. Cholecystokinin and leptin act synergistically to reduce body weight
36. Adaptation to high-fat diet reduces inhibition of gastric emptying by CCK and intestinal oleate
37. NMDA-type glutamate receptors participate in reduction of food intake following hindbrain melanocortin receptor activation
38. Frequency‐dependent facilitation of synaptic throughput via postsynaptic NMDA receptors in the nucleus of the solitary tract
39. DORSOMEDIAL HINDBRAIN PARTICIPATION IN CONTROL OF FOOD INTAKE
40. Preface
41. Diet-induced cholecystokinin release in cats
42. Circulating GLP-1 and CCK-8 reduce food intake by capsaicin-insensitive, nonvagal mechanisms
43. A tale of two endings: Modulation of satiation by NMDA receptors on or near central and peripheral vagal afferent terminals
44. Reduction of food intake by cholecystokinin requires activation of hindbrain NMDA-type glutamate receptors
45. Central Vagal Afferent Endings Mediate Reduction of Food Intake by Melanocortin-3/4 Receptor Agonist.
46. Vagal afferent neurons projecting to the stomach and small intestine exhibit multiple N-methyl-d-aspartate receptor subunit phenotypes
47. Modulation of vagal afferent excitation and reduction of food intake by leptin and cholecystokinin
48. Cholecystokinin1 receptors mediate the increase in Fos-like immunoreactivity in the rat myenteric plexus following intestinal oleate infusion
49. Reduced CCK-induced Fos expression in the hindbrain, nodose ganglia, and enteric neurons of rats lacking CCK-1 receptors
50. Increased food intake and CCK receptor antagonists: beyond abdominal vagal afferents
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