50 results on '"Winn, Michelle P"'
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2. Gq signaling causes glomerular injury by activating TRPC6
3. List of Contributors
4. Genetics and Chronic Kidney Disease
5. Pathogenesis and therapy of focal segmental glomerulosclerosis: an update
6. Arhgap24 inactivates Rac1 in mouse podocytes, and a mutant form is associated with familial focal segmental glomeruosclerosis
7. Exclusion of homozygous PLCE1 (NPHS3) mutations in 69 families with idiopathic and hereditary FSGS
8. OPINION: Genetic testing in nephrotic syndrome —challenges and opportunities
9. Focal and Segmental Glomerulosclerosis: Varying Biologic Mechanisms Underlie a Final Histopathologic End Point
10. TORCing up the Importance of Calcium Signaling
11. Screening for NPHS2 Mutations May Help Predict FSGS Recurrence after Transplantation
12. TRPC6 Enhances Angiotensin II-induced Albuminuria
13. Approach to the evaluation of heritable diseases and update on familial focal segmental glomerulosclerosis
14. Linkage of a Gene Causing Familial Membranoproliferative Glomerulonephritis Type III to Chromosome 1
15. Not All in the Family: Mutations of Podocin in Sporadic Steroid-Resistant Nephrotic Syndrome
16. Evidence for genetic factors in the development and progression of IgA nephropathy
17. Spectrum of disease in familial focal and segmental glomerulosclerosis
18. Orthostatic acute renal failure in a renal transplant
19. Clinical and genetic heterogeneity in familial focal segmental glomerulosclerosis
20. CAPÍTULO 19 - Causas Hereditárias de Síndrome Nefrótica
21. Variability in phenotype induced by the podocin variant R229Q plus a single pathogenic mutation
22. HLA-DQA1 and PLCG2 Are Candidate Risk Loci for Childhood-Onset Steroid-Sensitive Nephrotic Syndrome
23. A Novel Missense Mutation of Wilms’ Tumor 1 Causes Autosomal Dominant FSGS
24. Rare hereditary COL4A3/COL4A4 variants may be mistaken for familial focal segmental glomerulosclerosis
25. Mutations in the Gene That Encodes the F-Actin Binding Protein Anillin Cause FSGS
26. Phosphodiesterase 5 inhibition ameliorates angiontensin II-induced podocyte dysmotility via the protein kinase G-mediated downregulation of TRPC6 activity
27. TNXB Mutations Can Cause Vesicoureteral Reflux
28. COLABORADORES
29. Genetic testing in nephrotic syndrome—challenges and opportunities
30. A Hybrid CFHR3-1 Gene Causes Familial C3 Glomerulopathy
31. Inverted formin 2 mutations with variable expression in patients with sporadic and hereditary focal and segmental glomerulosclerosis
32. Hepatorenal Correction in Murine Glycogen Storage Disease Type I With a Double-stranded Adeno-associated Virus Vector
33. A New Locus for Familial FSGS on Chromosome 2P
34. Therapeutic targets in focal and segmental glomerulosclerosis
35. 2007 Young Investigator Award: TRP'ing into a New Era for Glomerular Disease
36. TRPC6 and FSGS: The latest TRP channelopathy
37. Unexpected Role ofTRPC6Channel in Familial Nephrotic Syndrome: Does It Have Clinical Implications?
38. Association of genetic polymorphisms with risk of renal injury after coronary bypass graft surgery
39. Parkin mutations and susceptibility alleles in late‐onset Parkinson's disease
40. Phosphodiesterase 5 inhibition ameliorates angiontensin II-induced podocyte dysmotility via the protein kinase G-mediated downregulation of TRPC6 activity.
41. Linkage of a Gene Causing Familial Focal Segmental Glomerulosclerosis to Chromosome 11 and Further Evidence of Genetic Heterogeneity
42. Focal segmental glomerulosclerosis: A need for caution in live-related renal transplantation
43. Modulation of the BP Response to Diet by Genes in the Renin-Angiotensin System and the Adrenergic Nervous System.
44. TRPC1 Channels Are Critical for Hypertrophic Signaling in the Heart.
45. Rare hereditary COL4A3/COL4A4 variants may be mistaken for familial focal segmental glomerulosclerosis
46. A new year to spread peace.
47. 112 Ang II Induces FAK Activation and Podocyte Migration Via a TRPC6-Dependent Mechanism
48. I'm the author of my own novel.
49. Gq signaling causes glomerular injury by activating TRPC6.
50. Unexpected role of TRPC6 channel in familial nephrotic syndrome: does it have clinical implications?
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