25 results on '"Thanabalasuriar, Ajitha"'
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2. Resident macrophages of the lung and liver: The guardians of our tissues
3. Bispecific antibody targets multiple pseudomonas aeruginosa evasion mechanisms in the lung vasculature
4. The inhibition of COPII trafficking is important for intestinal epithelial tight junction disruption during enteropathogenic Escherichia coli and Citrobacter rodentium infection
5. PD-L1 + neutrophils contribute to injury-induced infection susceptibility
6. Patrolling Alveolar Macrophages Conceal Bacteria from the Immune System to Maintain Homeostasis
7. The bacterial virulence factor NleAʼs involvement in intestinal tight junction disruption during enteropathogenic E. coli infection is independent of its putative PDZ binding domain
8. Dipeptidase-1 Is an Adhesion Receptor for Neutrophil Recruitment in Lungs and Liver
9. Rise and shine: Open your eyes to produce anti-inflammatory NETs
10. Neutrophil Extracellular Traps Confine Pseudomonas aeruginosa Ocular Biofilms and Restrict Brain Invasion
11. Unraveling the host's immune response to infection: Seeing is believing
12. PD-L1+ neutrophils contribute to injury-induced infection susceptibility.
13. α-Toxin Induces Platelet Aggregation and Liver Injury during Staphylococcus aureus Sepsis
14. Neonates, antibiotics and the microbiome
15. iNKT Cell Emigration out of the Lung Vasculature Requires Neutrophils and Monocyte-Derived Dendritic Cells in Inflammation
16. Molecular mechanisms of NET formation and degradation revealed by intravital imaging in the liver vasculature
17. Identification of Potentially Diarrheagenic Atypical Enteropathogenic Escherichia coli Strains Present in Canadian Food Animals at Slaughter and in Retail Meats
18. R-Spondin 2 signalling mediates susceptibility to fatal infectious diarrhoea
19. Sec24 interaction is essential for localization and virulence‐associated function of the bacterial effector protein NleA
20. The bacterial virulence factor NleA's involvement in intestinal tight junction disruption during EnteropathogenicE. coliinfection is independent of its putative PDZ binding domain
21. The bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by enteropathogenicEscherichia coli
22. Phosphorylated YDXV Motifs and Nck SH2/SH3 Adaptors Act Cooperatively To Induce Actin Reorganization
23. The Bacterial Virulence Factor NleA Inhibits Cellular Protein Secretion by Disrupting Mammalian COPII Function
24. The bacterial virulence factor NleA is required for the disruption of intestinal tight junctions by enteropathogenic Escherichia coli.
25. The bacterial virulence factor NleA's involvement in intestinal tight junction disruption during Enteropathogenic E. coliinfection is independent of its putative PDZ binding domain
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